Advances in Molecular Biology of Entamoeba histolytica
A special issue of Microorganisms (ISSN 2076-2607). This special issue belongs to the section "Medical Microbiology".
Deadline for manuscript submissions: 30 November 2025 | Viewed by 1041
Special Issue Editor
Interests: characterization of mRNA polyadenylation factors in E. histolytica; development of aptamers that recognize E. histolytica proteins
Special Issue Information
Dear Colleagues,
Entamoeba histolytica, the protozoan parasite responsible for human amoebiasis, affects up to 50 million people worldwide, causing about 26,000 deaths annually. This parasitic infection is endemic in developing countries, and it has also been reported in industrialized countries, mainly in returning travelers from endemic regions, men who have sex with men, and individuals living with HIV. Understanding the molecular basis of the biological activities of this pathogen has been a challenging topic for scientists. Knowledge of the parasite genome, application of recombinant DNA technology, use of omics data, development of new microenvironments in vitro, and bioinformatics approaches, among other methods, are providing new valuable information on various components of parasite molecular biology. This includes descriptions of genome organization, as well as the structure, function, and interactions of DNA, RNA, and proteins. Other aspects involve molecular mechanisms regulating gene expression, cellular processes, metabolic systems, host–pathogen interactions, relationships with the intestinal microbiome, pathogenicity, or drug resistance. This Special Issue aims to present the most recent scientific advances in the E. histolytica molecular biology that could contribute to advancements in the diagnosis and control of this human pathogen.
Dr. Laurence A. Marchat
Guest Editor
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Keywords
- Entamoeba histolytica
- genome structure and function
- protein structure and function
- pathogenicity and virulence
- bioinformatics
- omics
- relationships with host and microbiome
- metabolism
- gene expression
- drug resistance
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