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Life
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Effects of Psychiatric Disorders on Cardiology
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Effects of Psychiatric Disorders on Cardiology

A special issue of Life (ISSN 2075-1729). This special issue belongs to the section "Medical Research".

Deadline for manuscript submissions: closed (30 June 2023) | Viewed by 2846

Special Issue Editor

Dr. Cora Weber

E-Mail Website
Guest Editor
1. Abteilung Psychosomatische Medizin und Psychotherapie, Klinik Hennigsdorf, Oberhavel Kliniken GmbH, Marwitzer Str. 91, 16761 Hennigsdorf, Germany
2. Medical Clinic – Psychosomatic Medicine, Charité Universitätsmedizin Berlin, Campus Benjamin Franklin, Hindenburgdamm 30, 12200 Berlin, Germany
Interests: psychocardiology; stress; psychosomatic medicine; psychoneuroimmunology; cardiovascular disease

Special Issue Information

Dear Colleagues,

Research has demonstrated that both depression and anxiety are risk factors for coronary heart disease (CHD). Additionally, in heart failure, depression and exhaustion are majorly impactful, and call for therapeutic options.

There is a multi-system interaction of biological, psychological, and social factors involved in the pathogenesis of cardiovascular diseases.

Psychological and biological stress, neuroendocrine activity, genetic and epigenetic factors, procoagulant mechanisms, addiction, lifestyle—including diet and exercise—social factors, autonomic activity as indexed by heart-rate variability, and immune factors, including cytokines, all interact in the process of cardiovascular disease.

Relevant psychiatric disorders include depression, vital exhaustion, anxiety disorders, and trauma.

Therapeutic options include counseling methods, psychotherapy, collaborative care models and medication.

This Special Issue will collect papers written by international experts on cardiology, psychiatry, psychology, psychosomatic medicine and basic laboratory research.

Dr. Cora Stefanie Weber
Guest Editor

Manuscript Submission Information

Manuscripts should be submitted online at www.mdpi.com by registering and logging in to this website. Once you are registered, click here to go to the submission form. Manuscripts can be submitted until the deadline. All submissions that pass pre-check are peer-reviewed. Accepted papers will be published continuously in the journal (as soon as accepted) and will be listed together on the special issue website. Research articles, review articles as well as short communications are invited. For planned papers, a title and short abstract (about 100 words) can be sent to the Editorial Office for announcement on this website.

Submitted manuscripts should not have been published previously, nor be under consideration for publication elsewhere (except conference proceedings papers). All manuscripts are thoroughly refereed through a single-blind peer-review process. A guide for authors and other relevant information for submission of manuscripts is available on the Instructions for Authors page. Life is an international peer-reviewed open access monthly journal published by MDPI.

Please visit the Instructions for Authors page before submitting a manuscript. The Article Processing Charge (APC) for publication in this open access journal is 2600 CHF (Swiss Francs). Submitted papers should be well formatted and use good English. Authors may use MDPI's English editing service prior to publication or during author revisions.

Keywords

  • coronary heart disease
  • heart failure
  • depression
  • autonomic activity
  • heart-rate variability
  • coagulation
  • stress
  • genetic and epigenetic factors
  • cytokines
  • trauma

Published Papers (2 papers)

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Research

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11 pages, 441 KiB  
Article
Role of Heart Rate Variability in the Association between Myocardial Infarction Severity and Post-Myocardial Infarction Distress
by Reham Dyab, Claudia Zuccarella-Hackl, Mary Princip, Sinthujan Sivakumar, Rebecca E. Meister-Langraf, Hansjörg Znoj, Jean-Paul Schmid, Jürgen Barth, Ulrich Schnyder, Roland von Känel and Yori Gidron
Life 2023, 13(12), 2266; https://doi.org/10.3390/life13122266 - 27 Nov 2023
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Abstract
Objective: Myocardial infarction (MI) results in mental health consequences, including depression and post-traumatic stress disorder (PTSD). The risk and protective factors of such mental consequences are not fully understood. This study examined the relation between MI severity and future mental health consequences and [...] Read more.
Objective: Myocardial infarction (MI) results in mental health consequences, including depression and post-traumatic stress disorder (PTSD). The risk and protective factors of such mental consequences are not fully understood. This study examined the relation between MI severity and future mental health consequences and the moderating role of vagal nerve activity. Methods: In a reanalysis of data from the Myocardial Infarction-Stress Prevention Intervention (MI-SPRINT) study, 154 post-MI patients participated. MI severity was measured by the Killip Scale and by troponin levels. Depression and PTSD symptoms were assessed with valid questionnaires, both at 3 and 12 months. Vagal nerve activity was indexed by the heart rate variability (HRV) parameter of the root-mean square of successive R-R differences (RMSSD). Following multivariate analyses, the association between MI severity and distress was examined in patients with low and high HRV (RMSSD = 30 ms). Results: In the full sample, the Killip index predicted post-MI distress only at 3 months, while troponin predicted distress at 3- and 12-months post-MI. However, HRV moderated the effects of the Killip classification; Killip significantly predicted symptoms of depression and PTSD at 3- and 12-months post-MI, but only in patients with low HRV. Such moderation was absent for troponin. Conclusion: MI severity (Killip classification) predicted post-MI depression and PTSD symptoms, but only in patients with low HRV, suggesting that the vagal nerve is a partial protective (moderating) factor in the relation between Killip score and post-MI distress. Full article
(This article belongs to the Special Issue Effects of Psychiatric Disorders on Cardiology)
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16 pages, 731 KiB  
Systematic Review
The Role of Brain-Derived Neurotrophic Factor (BDNF) in Depression and Cardiovascular Disease: A Systematic Review
by Massimo Fioranelli, Maria Grazia Roccia, Bianca Przybylek and Maria Luisa Garo
Life 2023, 13(10), 1967; https://doi.org/10.3390/life13101967 - 26 Sep 2023
Cited by 2 | Viewed by 1858
Abstract
Background: Several studies have been conducted to prove the bidirectional relationship between cardiovascular disease (CVD) and depression. These two major illnesses share several common risk factors such that the development of either condition may increase the risk of the occurrence of the other. [...] Read more.
Background: Several studies have been conducted to prove the bidirectional relationship between cardiovascular disease (CVD) and depression. These two major illnesses share several common risk factors such that the development of either condition may increase the risk of the occurrence of the other. Brain-derived neurotrophic factor (BDNF) has been suggested as a reliable biomarker for depression and a strong predictor of CVD because it plays an important role in neuron survival and growth, serves as a neurotransmitter modulator, and promotes neuronal plasticity. The aim of this systematic review was to examine the bidirectional relationship between CVD and depression, focusing on the potential role of low serum BDNF levels in the development of either disease in the presence of the other. Methods: A systematic search strategy was developed using PRISMA guidelines. Results: Six studies (comprising 1251 patients) were identified, all of which examined the association between CVD and depression. Conclusions: It was found that there may be a strong association between low serum BDNF levels and the risk of post-stroke depression. However, the studies on the role of altered serum BDNF levels and other types of CVD are few. Therefore, the inverse association between depression and CVD cannot be proven. Full article
(This article belongs to the Special Issue Effects of Psychiatric Disorders on Cardiology)
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