State-of-Art in Autophagy and Neurodegeneration

Dear Colleagues, 

Autophagy suppression was shown in several neurodegenerative diseases, like Alzheimer's, Parkinson's, and Huntington's diseases connected with the accumulation of amyloid-β, α-synuclein, and huntingtin, respectively.

Autophagy is also reduced in aging, which associates with physiological dementia, neuronal integrity loss and increased susceptibility to neurodegenerative disorders including Alzheimer's Disease, Parkinson Disease and Frontotemporal dementia, to name a few.

Thus, the autophagy activation through different pathways can reverse both neuronal dopaminergic and behavioral deficits in vivo and seems to be a promising therapy in pathology. Induction of autophagy as a means of improving the viability of cells and organisms is an attractive approach to experimental therapy of neurodegeneration.

This Special Issue of Cells will present research articles and reviews that cover the scope of autophagy and neurodegeneration. All scientists working in these fields are cordially invited to submit their manuscripts.

Prof. Dr. Ciro Isidoro
Prof. Dr. Tatyana A. Korolenko
Guest Editors

Manuscript Submission Information

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