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The Molecular Research on Incretins and Diabetic Comorbidities
This topical collection belongs to the section “Cellular Metabolism“.
Topical Collection Information
Dear Colleagues,
Diabetes increases the risk of various comorbidities, such as coronary artery disease, peripheral artery disease, retinopathy, neuropathy, nephropathy, and stroke in patients with diabetes. Glucose-dependent insulinotropic polypeptide (GIP) and glucagon-like peptide-1 (GLP-1) could ameliorate hyperglycemia and prevent the related comorbidities in patients with diabetes. However, the molecular mechanisms of GIP or GLP-1 on the diabetic comorbidities remain to be elucidated.
For this Collection, we invite original research and review articles on recent progress in molecular mechanisms of incretins and diabetic complications in animals or humans, in vitro culture experiments, and in vivo models to investigate the potential of incretins to the comorbidities in diabetes.
Dr. Michishige Terasaki
Collection Editor
Manuscript Submission Information
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Submitted manuscripts should not have been published previously, nor be under consideration for publication elsewhere (except conference proceedings papers). All manuscripts are thoroughly refereed through a single-blind peer-review process. A guide for authors and other relevant information for submission of manuscripts is available on the Instructions for Authors page. Cells is an international peer-reviewed open access semimonthly journal published by MDPI.
Please visit the Instructions for Authors page before submitting a manuscript. The Article Processing Charge (APC) for publication in this open access journal is 2700 CHF (Swiss Francs). Submitted papers should be well formatted and use good English. Authors may use MDPI's English editing service prior to publication or during author revisions.
Keywords
- GIP
- GLP-1
- differentiation
- atherosclerosis
- intestine
- animal models
- drug screening

