Molecular Advances and New Therapeutic Approaches in Endometriosis

Dear Colleagues,

The human endometrium is unique because it is a highly regenerative tissue and heals without scarring. Menstruation generates not only pain but also, via retrograde menstruation, contributes to endometriosis. Although Sampson’s implantation hypothesis is generally accepted as the most likely cause of endometriosis, more hypotheses, such as inflammation, hypoxia etc., are needed to explain why only a minor fraction of women get the disease.

The aim of this Special Issue is to highlight molecular pathways involved in endometriosis that can help to develop or improve new and existing therapies, especially with respect to pain. The main focus will be on in vitro and animal models.

Areas to be included in this Special Issue are focused on but not limited to: molecular pathways, new therapies, pathogenesis, stem cells, stromal/mesenchymal cells, epithelial cells, epithelial–mesenchymal transition, migration, invasion, pain, in vitro models, animal model organoids, 3D models and architectures, non-invasive diagnostics, next-generation single cell sequencing, and array analysis.

We hope that this Special Issue will broaden and deepen our knowledge on endometriosis with respect to the topics mentioned above, and that it will be of interest to both scientists and clinicians. Thus, contributions by experts in the field of endometriosis, in the form of original research articles and/or reviews, are most welcome. 

Dr. Lutz Konrad
Guest Editor

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