Lipid Peroxidation and Protein Carbonylation in Human Diseases
A special issue of International Journal of Molecular Sciences (ISSN 1422-0067). This special issue belongs to the section "Biochemistry".
Deadline for manuscript submissions: 20 June 2025 | Viewed by 2100
Special Issue Editor
Interests: oxidative stress; diabetes; natural antioxidants; asthma; free radicals
Special Issues, Collections and Topics in MDPI journals
Special Issue Information
Dear Colleagues,
Free radicals, whether produced in the human body during metabolic processes or derived from the environment, have the potential to oxidize both lipids and proteins. This oxidation process can lead to the formation of lipid peroxides and oxidized forms of proteins that play critical roles in cell death, including apoptosis, autophagy, and ferroptosis. The body has built-in defense mechanisms to counteract the harmful effects of free radicals, including the presence of antioxidants, such as vitamins C and E, and enzymes, such as superoxide dismutase and catalase. These antioxidants donate electrons to free radicals, neutralizing their reactivity and preventing further oxidative damage. When there is an imbalance between free radical production and the body’s antioxidant defenses, oxidative stress can occur. Oxidative stress is associated with a wide range of health problems, including ageing, neurodegenerative diseases, cardiovascular diseases, cancer, and inflammatory diseases.
Dr. Galina Nikolova
Guest Editor
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Keywords
- free radicals
- lipid peroxidation
- protein carbonylation
- apoptosis
- autophagy
- ferroptosis
- antioxidant enzymes
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