Advanced Communications in Cardiovascular Disease: Small Vesicles and Cell Identity Theft

Dear Colleagues,

The interactions between metabolic organs have evolved concomitantly to maintain whole-body energy homeostasis and ensure the organism’s adaption to external cues. Extracellular vesicles and intracellular vesicles are emerging as a new category of messengers that facilitate crosstalk among organs and within cells. Dynamic movement of the small vesicles is the basis of maintaining internal environment stabilization. Small vesicles play a pivotal role in various cardiovascular diseases, including diabetic cardiomyopathy. Macroautophagy (classic autophagy) in particular is an evolutionarily conserved mechanism for the maintenance of physiological homeostasis and balancing energies in reusage and expenditure. As energy resources, mitochondria are essential to cardiovascular homeostasis in health. Maintaining a healthy mitochondrial balance and rebalancing dysfunctional mitochondria quality and quantity are involved in various therapeutic strategies. 

In order to develop new insights into the intimate connection between small vesicles, including small extracellular vesicles, and intracellular vesicle movement in cardiovascular disorders, we will introduce the genetic and epigenetic role of regulatory factors and maneuvers that alter extra-/intracellular communication in various types of cardiomyopathy and in cardiovascular diseases.

This Special Issue entitled “Advanced Communications in Cardiovascular Disease: Small Vesicles and Cell Identity Ttheft” welcomes manuscripts from original investigations and comprehensive reviews demonstrating new and advanced research viewpoints on the mechanisms of regulating inter-/intracellular communications to cardiovascular disease with or without diabetes.

Dr. Yajing Wang
Prof. Dr. William Chilian
Guest Editors

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