Journal of Fungi, Volume 12, Issue 9
2026 September - 80 articles
Cover Story: The rise in multi-drug-resistant fungal pathogens exposes the limits of conventional antifungals and emphasizes the need for alternative biocontrols. Saccharomycopsis schoenii, a CUG-Ser2 clade yeast, has emerged as a model for studying contact-dependent necrotrophy. Its obligate predatory lifestyle is driven by extensive genomic restructuring, including the loss of the sulfate assimilation pathway, which links starvation to a predatory switch. To breach prey cell walls, S. schoenii takes a combined approach involving rewired MAPK signaling, penetration pegs, and tandem expansions of both secreted aspartic proteases (SAPs) and carbohydrate-active enzymes (CAZymes). This review highlights recent genomic and functional studies to explain the evolutionary basis of this behavior. View this paper
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J. Fungi - ISSN 2309-608X