In the original publication [1], there was a mistake in Figure 7 as published. During the preparation of the manuscript, a draft copy of the figure was inadvertently uploaded instead of the finalized figure intended for publication. The authors apologize for this inconvenience. The corrected Figure 7 appears below. The authors state that the scientific conclusions are unaffected. This correction was approved by the Academic Editor. The original publication has also been updated.
Figure 7.
Retinal ganglion cell (RGC) density. (A) Representative image of a control flat mount immunostained for the brain-specific homeobox/POU domain protein 3A (Brn3a). (B–H) Representative high-magnification images from the middle retina (sampling location at about 2.5 mm from the optic disc) from control rats (B) or rats that received MCE either untreated (C) or treated with vehicles (D,E), timolol (F), brimonidine (G) or melatonin/agomelatine (H). Scale bars: 1 mm (A) or 150 µm (B–H). Magnification: 4× (A) or 20× (B–H). (I) RGC density based on counting analysis of Brn3a-labeled cells. MCE injection reduced RGC density. Decreased RGC density was unaffected by vehicles, but partially recovered with timolol. After brimonidine or melatonin/agomelatine, RGC density recovered to its control value. Data are shown as box-and-whiskers plots (n = 6 retinas for each experimental group). * p < 0.05 versus control; § p < 0.05 versus the respective vehicle (one-way ANOVA followed by Tukey’s post hoc test). MelAgo—melatonin/agomelatine.
Reference
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