The Pathogenetic Triad of Chronic Airway Diseases—Inflammation, Tissue Destruction, and Airway Remodeling
A special issue of Cells (ISSN 2073-4409). This special issue belongs to the section "Cellular Immunology".
Deadline for manuscript submissions: 15 November 2024 | Viewed by 1678
Special Issue Editors
Interests: airway diseases
Special Issues, Collections and Topics in MDPI journals
Special Issue Information
Dear Colleagues,
Chronic respiratory diseases such as asthma, COPD, idiopathic pulmonary fibrosis, and cystic fibrosis affect hundreds of millions of people worldwide and are associated with a significant loss of quality of life and high mortality. A significant impairment of lung function is central to these diseases and arises from a pathogenetic triad of chronic inflammation, tissue destruction, and airway remodeling. Since chronic lung diseases cannot be cured and, at best, only their symptoms can be alleviated, it is extremely important to gain further knowledge about the underlying pathomechanisms. New methods such as 3D-organoid cultures, single-cell sequencing, spatial transcriptomics, and single-cell epigenomics have the potential to pave the way for a deeper understanding of chronic lung diseases and the identification of novel therapeutic approaches.
Here, we would like to offer a platform for preclinical and clinical research results that address the complexity of chronic respiratory diseases. We encourage the submission of original research articles, reviews, or methodological articles dealing with various aspects of airway diseases. Contributions may address, but are not limited to, cellular mechanisms underlying inflammation, tissue destruction, and airway remodeling. Studies with clear translational potential are especially welcome.
Prof. Dr. Christoph Beisswenger
Dr. Michael Wegmann
Guest Editors
Manuscript Submission Information
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Keywords
- chronic lung disease
- inflammation
- signaling pathway
- disease models
- tissue destruction
- airway remodeling
- epigenetics
