Editorial Board Members’ Collection Series: Physiology and Drug Discovery of Neurodegenerative Diseases
A special issue of Biology (ISSN 2079-7737).
Deadline for manuscript submissions: closed (30 November 2023) | Viewed by 12693
Special Issue Editors
2. Lineberger Comprehensive Cancer Center, The University of North Carolina at Chapel Hill, Chapel Hill, NC 27599, USA
Interests: X-ray protein crystallography; enzymology; structure-based drug design
Interests: enzymology; protein chemistry; transglutaminase; gene regulation; oxidative damage
Special Issue Information
Dear Colleagues,
We are pleased to invite you to contribute your work to a Special Issue of Biology entitled “Editorial Board Members’ Collection Series: Physiology and Drug Discovery of Neurodegenerative Diseases”. Neurodegenerative diseases are caused by the progressive loss of neuron structure in the process of neurodegeneration, which ultimately leads to neuron cell death. The major neurodegenerative diseases include Alzheimer’s disease, Parkinson’s disease, amyotrophic lateral sclerosis, multiple sclerosis, Huntington’s disease, multiple-system atrophy, prion diseases, and Friedreich’s ataxia. Since there are currently no methods or treatments that can effectively reverse the progressive degeneration of neurons, these diseases are considered incurable.
The aim of this Special Issue is to collect articles that represent the current knowledge and progress in neurodegenerative diseases for dissemination to the readers, and to promote the progress of the field. In this Special Issue, original research articles and reviews are welcome. Although Alzheimer’s disease, Parkinson’s disease, and amyotrophic lateral sclerosis are the major focuses of this Special Issue, any articles on neurodegenerative diseases are welcome.
We look forward to receiving your contributions.
Prof. Dr. Hengming Ke
Prof. Dr. Carlo M. Bergamini
Guest Editors
Manuscript Submission Information
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Keywords
- neurodegenerative diseases
- Alzheimer’s
- Parkinson’s
- Huntington’s
- amyotrophic lateral sclerosis
- multiple-system atrophy
- prion diseases
- Friedreich’s ataxia
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