Moriyama, N.; Saito, M.; Ono, Y.; Yamashita, K.; Aoi, T.; Kotani, J.
Increased Interleukin-17-Producing γδT Cells in the Brain Exacerbate the Pathogenesis of Sepsis-Associated Encephalopathy and Sepsis-Induced Anxiety in Mice. J. Clin. Med. 2023, 12, 4309.
https://doi.org/10.3390/jcm12134309
AMA Style
Moriyama N, Saito M, Ono Y, Yamashita K, Aoi T, Kotani J.
Increased Interleukin-17-Producing γδT Cells in the Brain Exacerbate the Pathogenesis of Sepsis-Associated Encephalopathy and Sepsis-Induced Anxiety in Mice. Journal of Clinical Medicine. 2023; 12(13):4309.
https://doi.org/10.3390/jcm12134309
Chicago/Turabian Style
Moriyama, Naoki, Masafumi Saito, Yuko Ono, Kimihiro Yamashita, Takashi Aoi, and Joji Kotani.
2023. "Increased Interleukin-17-Producing γδT Cells in the Brain Exacerbate the Pathogenesis of Sepsis-Associated Encephalopathy and Sepsis-Induced Anxiety in Mice" Journal of Clinical Medicine 12, no. 13: 4309.
https://doi.org/10.3390/jcm12134309
APA Style
Moriyama, N., Saito, M., Ono, Y., Yamashita, K., Aoi, T., & Kotani, J.
(2023). Increased Interleukin-17-Producing γδT Cells in the Brain Exacerbate the Pathogenesis of Sepsis-Associated Encephalopathy and Sepsis-Induced Anxiety in Mice. Journal of Clinical Medicine, 12(13), 4309.
https://doi.org/10.3390/jcm12134309