Common Mechanisms in Gestational Diabetes, Preeclampsia, and Coronavirus Disease 2019

Dear Colleagues,

The leading cause of changes in the placenta is inflammation that is characterized by immunoglobulins, C-reactive protein, interleukins, and cytokines TNF-α and IFN-γ, as well as decidual cytokine IL-32.

We are pleased to invite you to present your results in the field of low-grade inflammation, immunoglobulins, glycolisation, platelet aggregation, and microvascular thrombosis. Inflammation induces tissue damage that can be measured with inflammation markers, immunoglobulins, glycolisation, platelet aggregation, and microvascular thrombosis.

The pro-angiogenic effects on the fetoplacental circulation support trophoblast growth, and placental angiogenesis will be monitored by measuring serum vascular endothelial growth factor (VEGF), Annexin A2, and placental growth factor (PGF). Also, it is required to investigate umbilical cord plasma sclerostin as a predictor of placental and newborn weight.

Suggested themes and article types for submissions: original research (brief reports, research letters), review articles (narrative, systematic, meta-analysis, clinical practice, epidemiology), case reports, and methodology articles. 

Research areas may include (but are not limited to) the following:

• A high-risk pregnancy.
• Hormonal and physiological changes that cause insulin resistance in GDM.
• Gestational hypertension, preeclampsia, and eclampsia in coronavirus disease 2019.
• Low-grade inflammation.
• Glycolisation of immunoglobulins during GDM, PE, and COVID-19.
• Placental angiogenesis.
• Change in platelet aggregation during pregnancy.
• Change in mother milk composition due to nutritional factors in GDM and PE.

We look forward to receiving your contributions.

Dr. Sandor G. Vari
Guest Editor

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