Mitochondrial Dysfunction in Degenerative Diseases
A special issue of Cells (ISSN 2073-4409). This special issue belongs to the section "Mitochondria".
Deadline for manuscript submissions: closed (20 July 2021) | Viewed by 1720
Special Issue Editor
2. Department of Chemical & Systems Biology, Stanford University School of Medicine, Stanford, CA, USA
Interests: bioenergetics; mitochondrial proteostasis; aldehyde metabolism; cardiac and skeletal muscle diseases; therapeutics
Special Issue Information
Dear Colleagues,
Mitochondrial dysfunction is critical in the establishment and progression of degenerative diseases. Therefore, mechanisms of surveillance and quality control capable of maintaining mitochondrial integrity and functionality are critical for cellular homeostasis. Mitochondrial quality is controlled by a myriad of interconnected systems, including: 1. enzymatic and non-enzymatic elements capable of fighting oxygen-mediated mitochondrial toxicity; 2. mitochondrial proteases and chaperones responsible for the maintenance of mitochondrial proteostasis; and 3. a multilayer network of proteins involved in the control of mitochondrial morphology, location, and number. In this Special Issue, we invite investigators to contribute their original research, perspective or review articles to broaden our understanding of the regulatory processes involved in mitochondrial bioenergetics, surveillance, and quality control mechanisms in health and disease. We encourage the submission of basic, translational, and clinical studies describing signaling mechanisms and novel approaches for diagnostics and therapeutics according to the proposed topic.
Dr. Julio Cesar Batista Ferreira
Guest Editor
Manuscript Submission Information
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Keywords
- redox signaling and oxidative stress
- mitochondrial proteostasis
- mitochondria-nucleus communication (mitochondrial retrograde signaling)
- mitochondrial dynamics: mitochondrial biogenesis, fusion, and fission
- mitochondrial clearance (mitophagy)
- mitochondrial bioenergetics
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