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Nutrients
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Nutrition, Obesity, and Chronic Diseases: The Link Between Nutrition and...
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Nutrition, Obesity, and Chronic Diseases: The Link Between Nutrition and Obesity-Related Diseases

A special issue of Nutrients (ISSN 2072-6643). This special issue belongs to the section "Nutrition and Public Health".

Deadline for manuscript submissions: closed (25 October 2025) | Viewed by 1492

Special Issue Editor

Prof. Dr. Hae-Jin Ko

E-Mail Website
Guest Editor
Department of Family Medicine, School of Medicine, Kyungpook National University, Daegu 41944, Republic of Korea
Interests: obesity; metabolic disorders; primary healthcare; family medicine; chronic diseases; cancer screening
Special Issues, Collections and Topics in MDPI journals

Special Issue Information

Dear Colleagues,

This Special Issue of the journal Nutrients focuses on the critical intersections between nutrition, obesity, and chronic diseases, examining how dietary factors influence the prevalence and progression of obesity-related health conditions. Nutritional interventions serve as a pivotal treatment approach, with their significance greatly enhanced when integrated with other therapies such as pharmacological and surgical treatments. In a world where chronic diseases such as diabetes, hypertension, cardiovascular disease, and fatty liver disease continue to rise, understanding the role of nutrition in managing obesity is more crucial than ever.

This Special Issue aims to bring together a range of research articles, reviews, and that explore various aspects of how nutrition impacts obesity and its associated health risks. From the efficacy of personalized dietary interventions to the socio-economic factors influencing dietary choices, this Special Issue will shed light on the complex relationships between diet, body weight, and chronic disease outcomes.

Through rigorous research and thoughtful discussion, this Special Issue seeks to inform and inspire healthcare professionals, researchers, and policymakers to develop more effective strategies for combating obesity and enhancing public health.

Prof. Dr. Hae-Jin Ko
Guest Editor

Manuscript Submission Information

Manuscripts should be submitted online at www.mdpi.com by registering and logging in to this website. Once you are registered, click here to go to the submission form. Manuscripts can be submitted until the deadline. All submissions that pass pre-check are peer-reviewed. Accepted papers will be published continuously in the journal (as soon as accepted) and will be listed together on the special issue website. Research articles, review articles as well as short communications are invited. For planned papers, a title and short abstract (about 250 words) can be sent to the Editorial Office for assessment.

Submitted manuscripts should not have been published previously, nor be under consideration for publication elsewhere (except conference proceedings papers). All manuscripts are thoroughly refereed through a single-blind peer-review process. A guide for authors and other relevant information for submission of manuscripts is available on the Instructions for Authors page. Nutrients is an international peer-reviewed open access semimonthly journal published by MDPI.

Please visit the Instructions for Authors page before submitting a manuscript. The Article Processing Charge (APC) for publication in this open access journal is 2900 CHF (Swiss Francs). Submitted papers should be well formatted and use good English. Authors may use MDPI's English editing service prior to publication or during author revisions.

Keywords

  • nutrition
  • obesity
  • overweight
  • chronic disease
  • obesity-related disease
  • diabetes
  • insulin resistance
  • hypertension
  • cardiovascular disease
  • metabolic dysfunction-associated steatotic liver disease
  • diet

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Further information on MDPI's Special Issue policies can be found here.

Published Papers (1 paper)

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Research

17 pages, 3786 KB  
Article
Effects of Blue Light Exposure on Hepatic Inflammation and Gut Microbiota in Mice Consuming a High-Fat, High-Fructose Diet
by Wen-Chih Huang, Pei-Ni Lee, Wan-Ju Yeh, Wen-Chi Wu, Hsin-Yu Shih, Yi-Jen Chen and Hsin-Yi Yang
Nutrients 2026, 18(1), 164; https://doi.org/10.3390/nu18010164 - 4 Jan 2026
Viewed by 958
Abstract
Background: High-fat or high-fructose consumption may cause abnormal lipid accumulation in the liver, resulting in fatty liver disease, and the intervention of other stress factors may accelerate the progression of this condition. Many studies have demonstrated that long-term exposure to blue light may [...] Read more.
Background: High-fat or high-fructose consumption may cause abnormal lipid accumulation in the liver, resulting in fatty liver disease, and the intervention of other stress factors may accelerate the progression of this condition. Many studies have demonstrated that long-term exposure to blue light may not only injure the eyes but also cause an increase in oxidative stress, which has been related to metabolic and gut microbiota disorders. However, current research on whether blue light exposure exacerbates fatty liver disease still remains limited. Objective: Therefore, the aim of this study is to investigate the effect of a high-fat, high-fructose diet combined with blue light exposure on fatty liver disease progression. Method: In the first part of the study, we observed that 16 weeks of blue light exposure alone did not achieve significant effects in the liver of male, female, or OVX mice. Therefore, in the second part, we fed ICR mice a high-fat, high-fructose (HFHF) diet to investigate the effect of simultaneous 16-week exposure to blue light. The mice were assigned to three groups, control group (C), HFHF diet group (H), and HFHF diet plus blue light exposure group (HB), to investigate the intervention of unhealthy diet composition and blue light exposure on hepatic oxidative and inflammatory makers and gut microbiota composition. Results: The results showed that exposure to blue light exacerbates oxidative stress (hepatic MDA, p < 0.009), and inflammatory damage (lobular inflammation score, p < 0.0001; hepatic TNF-α, p = 0.0074) caused by an HFHF diet, but this mechanism is not mediated by the TLR4 signaling pathway. Furthermore, exposure to blue light may also partially affect the composition of the gut microbiota. Conclusions: The results of the study suggested that under unhealthy dietary conditions, long-term blue light exposure may be one of the risk factors accelerating the progression of fatty liver disease. Full article
(This article belongs to the Special Issue Nutrition, Obesity, and Chronic Diseases: The Link Between Nutrition and Obesity-Related Diseases)
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