Women’s Special Issue Series: Livers—Honouring Women in Hepatology

A special issue of Livers (ISSN 2673-4389).

Deadline for manuscript submissions: closed (31 October 2023) | Viewed by 552

Special Issue Editor


E-Mail Website
Guest Editor
Institute of Experimental Immunology, University Bonn, Venusberg Campus 1, 53127 Bonn, Germany
Interests: NASH; liver dendritic cells; immunmodulation; stroll cells; progenitor cells

Special Issue Information

Dear Colleagues, 

The liver as an immune milieu.

The liver is our metabolic center which also plays a complex role in immunity and tolerance.

The liver parenchyma is rich in a variety of immune cells involving Kupffer cells, monocytes, dendritic cells, innate (NK, NKT cells and γδT cells) and classical lymphocytes (-T and B cells). All of these immune cells interact with liver parenchymal cells to create a specific microenvironment with complex regulatory circuits that maintain liver homeostasis. Perturbance in these circuits results in various liver diseases such as nonalcoholic liver steatohepatitis, and autoimmune-, or cholestatic liver diseases. Chronic imbalances in liver homeostasis often lead ultimately to fibrosis and/or cancer development which makes it especially intriguing to understand the pathomechanism and find novel therapeutic approaches.

In this issue, we would like to welcome articles in every field of liver biology specifically focusing on the liver as an immune milieu.

This Special Issue aims to give visibility to research works where the lead author is a woman (i.e., a person who identifies as a woman) or that are completely authored by women.

Although we encourage women scientists to submit original or review manuscripts to this Special Issue, we strongly promote a policy of gender equality and welcome submissions from all authors, irrespective of gender.

Prof. Dr. Veronika Lukacs-Kornek
Guest Editor

Women’s Special Issue Series

This Special Issue is part of Livers's Women’s Special Issue Series, hosted by women editors for women researchers. The Series advocates the advancement of women in science. We invite contributions to the Special Issue whose lead authors identify as women. The submission of articles with all-women authorship is especially encouraged. However, we do welcome articles from all authors, irrespective of gender.

Manuscript Submission Information

Manuscripts should be submitted online at www.mdpi.com by registering and logging in to this website. Once you are registered, click here to go to the submission form. Manuscripts can be submitted until the deadline. All submissions that pass pre-check are peer-reviewed. Accepted papers will be published continuously in the journal (as soon as accepted) and will be listed together on the special issue website. Research articles, review articles as well as short communications are invited. For planned papers, a title and short abstract (about 100 words) can be sent to the Editorial Office for announcement on this website.

Submitted manuscripts should not have been published previously, nor be under consideration for publication elsewhere (except conference proceedings papers). All manuscripts are thoroughly refereed through a single-blind peer-review process. A guide for authors and other relevant information for submission of manuscripts is available on the Instructions for Authors page. Livers is an international peer-reviewed open access quarterly journal published by MDPI.

Please visit the Instructions for Authors page before submitting a manuscript. The Article Processing Charge (APC) for publication in this open access journal is 1000 CHF (Swiss Francs). Submitted papers should be well formatted and use good English. Authors may use MDPI's English editing service prior to publication or during author revisions.

Keywords

  • liver
  • immune milieu
  • liver parenchyma
  • immune cells
  • liver diseases
  • nonalcoholic liver steatohepatitis
  • autoimmune- or cholestatic liver diseases
  • women
  • female

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Published Papers

There is no accepted submissions to this special issue at this moment.
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