Cellular and Molecular Mechanisms of Idiopathic Pulmonary Fibrosis
A special issue of Cells (ISSN 2073-4409).
Deadline for manuscript submissions: 20 April 2026 | Viewed by 13
Special Issue Editors
Interests: molecular biology; lung disease; drug discovery; research mechanism in respiratory disease
2. Toronto General Hospital Research Institute, University Health Network, 101 College Street PMCRT 2-814, Toronto, ON M5G 1L7, Canada
Interests: acute lung injury; lung transplantation; drug discovery; nano-drug formulation; signal transduction; cell biology; thyroid function
Special Issue Information
Dear Colleagues,
Idiopathic pulmonary fibrosis (IPF) is a progressive and fatal interstitial lung disease characterized by aberrant epithelial repair, fibroblast activation, immune dysregulation, and excessive extracellular matrix deposition. Although recent studies have advanced our understanding of IPF, the molecular and cellular mechanisms underlying disease initiation and progression remain incompletely defined, and effective therapeutic options are still limited.
This Special Issue, entitled “Cellular and Molecular Mechanisms of Idiopathic Pulmonary Fibrosis”, will provide a comprehensive platform to explore the fundamental biology of IPF within the scope of Cells. We invite submissions focusing on the pathways and processes regulating epithelial–mesenchymal crosstalk, immune and inflammatory responses, genetic and epigenetic regulation, metabolic reprogramming, cellular senescence, autophagy, and extracellular matrix remodeling. Both mechanistic studies and integrative approaches that identify novel biomarkers, therapeutic targets, or innovative models are welcome.
In this Special Issue, original research articles and reviews are encouraged. Research areas may include, but are not limited to, cellular and molecular signaling, disease modeling, and translational strategies that deepen our understanding of IPF biology. We look forward to receiving your contributions.
Dr. Sooyeon Lee
Prof. Dr. Mingyao Liu
Guest Editors
Manuscript Submission Information
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Keywords
- idiopathic pulmonary fibrosis
- fibroblast activation
- epithelial–mesenchymal interaction
- extracellular matrix remodeling
- inflammation
- signaling pathways
- biomarkers
- molecular targets
- autophagy
- senescence
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