Molecular and Cellular Mechanisms in Cardiovascular Dysfunction and Toxicity
A special issue of Cells (ISSN 2073-4409). This special issue belongs to the section "Cells of the Cardiovascular System".
Deadline for manuscript submissions: closed (30 April 2023) | Viewed by 5176
Special Issue Editors
Interests: cardiac dysfunction; bio-therapy; cardio-oncology; biomarker; pathophysiology; cardio-circulatory failure
Interests: cardiac dysfunction; metabolism; oxidative stress; post-translational modification; pathophysiology
Special Issue Information
Dear Colleagues,
Cardiovascular diseases are a leading cause of death worldwide. This issue will focus on known and new, shared and specific, signaling pathways leading to the large spectrum of cardiovascular diseases among which are heart failure, myocardial infarction, hypertension and genetic or reversible cardiomyopathies. Mechanisms of cardiac toxicity occurring as a side effect of chemotherapy and radiotherapy will also be highlighted in this Special Issue.
This Special Issue deals with abnormalities in cardiomyocytes contraction and relaxation including calcium handling, mitochondria and energy homeostasis, oxidative stress and cardiac cells survival and apoptosis, extracellular matrix, cell micro-environment and cell-cell crosstalk. Studies on alteration of hormonal and neurohormonal signaling including adrenergic system, renin–angiotensin–aldosterone system alongside with sex hormones leading to cardiac dysfunction or toxicity will be included in this issue.
This Special Issue aims to provide a better understanding of molecular and cellular mechanisms leading to cardiac damage, that are still not well understood. Furthermore, our aim is to gain a general overview of known and new therapeutic approaches and drug targets in the field.
Dr. Feriel Azibani
Dr. Emilie Dubois-Deruy
Guest Editors
Manuscript Submission Information
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Keywords
- mitochondria
- oxidative stress
- chemotherapy
- cell survival
- cytokines
- cardiomyocyte
- fibrosis
- calcium handling
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