Exploring the Molecular and Cellular Mechanisms of Pulmonary Arterial Hypertension
A special issue of Biomolecules (ISSN 2218-273X). This special issue belongs to the section "Molecular Medicine".
Deadline for manuscript submissions: 31 March 2026 | Viewed by 19
Special Issue Editors
Interests: ion channels; Ca2+ channels; K+ channels; electrophysiology; Patch-clamp recording; pulmonary hypertension; vascular cells; RV dysfunction; cardiomyocytes; arterial tone
Special Issues, Collections and Topics in MDPI journals
Special Issue Information
Dear Colleagues,
Pulmonary arterial hypertension (PAH) is a rare and life-threatening disease characterized by a multifactorial pathobiology involving all major cell types of the pulmonary circulation, including pulmonary arterial smooth muscle cells, endothelial cells, adventitial fibroblasts, and right ventricular (RV) cardiomyocytes. PAH has a poor prognosis and ultimately leads to right ventricular failure (RVF), underscoring the urgent need for innovative therapeutic strategies.
This Special Issue focuses on exploring the cellular mechanisms and molecular aspects of pulmonary arterial hypertension in the pulmonary vasculature and right ventricular cardiomyocytes, including PAH-predisposing genes, ion channel remodeling, transcription factors, and RNA-binding proteins. We kindly welcome submissions, including original papers and reviews, on this widely discussed topic.
Dr. Fabrice Antigny
Dr. Grégoire Ruffenach
Guest Editors
Manuscript Submission Information
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Keywords
- pulmonary arterial hypertension
- PAH predisposing genes
- ion channels
- transcription factors
- RNA-binding protein
- omics approaches
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