Oxidative Stress in Neurodegeneration
A special issue of Antioxidants (ISSN 2076-3921). This special issue belongs to the section "Health Outcomes of Antioxidants and Oxidative Stress".
Deadline for manuscript submissions: closed (10 October 2022) | Viewed by 35681
Special Issue Editors
Interests: neurodegenerative acute (traumatic brain injury) and chronic (multiple sclerosis) disorders; oxidative and nitrosative stresses; oxidation mechanisms; antioxidants; metabolomics; biochemical analytical techniques; lipid peroxidation products; dietary antioxidants; carotenoids; polyphenols; vitamin E; vitamin C; antioxidant activity/capacity
Special Issues, Collections and Topics in MDPI journals
Interests: mitochondrial dysfunction; oxidative/nitrosative stress; neurodegenerations; multiple sclerosis; traumatic brain injury; metabolic cell reprogramming
Special Issues, Collections and Topics in MDPI journals
Special Issue Information
Dear Colleagues,
Carrying out a search on PubMed for “Oxidative Stress + neurodegeneration” on 16 February 2022, we found that 7953 full-length papers have been published to that date. This highlights the central role displayed by oxidative stress in the pathobiological mechanisms of development and progression of acute (stroke, traumatic brain injury) and chronic (amyotrophic lateral sclerosis, multiple sclerosis, Alzheimer’s disease, Parkinson’s disease, etc.) neurodegenerations. A better understanding of biochemical/metabolic/molecular processes involving the raising of oxidative stress in acute and chronic neurodegenerations may represent the main road to increasing our knowledge regarding the molecular mechanisms responsible for cellular malfunctioning and also for the development of new effective pharmacological approaches.
The goal of this Special Issue is to provide new advances connecting alterations of the pattern of antioxidants and scavenger compounds, mitochondrial dysfunction, imbalance of energy metabolism, and oxidative stress with onset and/or progression of acute and chronic neurodegenerations, either obtained in experimental cellular/animal models or in clinical studies. Papers reporting data on new drug treatments aiming to reduce or avoid oxidative stress are highly welcome. Reviews highlighting the most recent information in specific acute or chronic neurodegenerative disease will also be taken into consideration.
Prof. Dr. Barbara Tavazzi
Prof. Dr. Giacomo Lazzarino
Guest Editors
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