nutrients-logo

Journal Browser

Journal Browser

Impact of Physical Activity, Diet Quality on Cardiometabolic Health

A special issue of Nutrients (ISSN 2072-6643). This special issue belongs to the section "Sports Nutrition".

Deadline for manuscript submissions: closed (15 January 2024) | Viewed by 3676

Special Issue Editors


E-Mail Website
Guest Editor
Department of Physiology, Wayne State University, Detroit, MI, USA
Interests: cardiovascular disease; salt sensitive hypertension; exercise training
Special Issues, Collections and Topics in MDPI journals

E-Mail Website
Guest Editor
Department of Physiology, Wayne State University, Detroit, MI, USA
Interests: neural control of cardiovascular function; heart failure; hypertension; exercise

Special Issue Information

Dear Colleagues,

The goal of this Special Issue, “Impact of Physical activity, Diet quality on Cardiometabolic Health”, is to focus on the importance of physical activity and/or dietary consumption cardiovascular and metabolic health.

Specifically, the aim is to identify the mechanisms and impact of sedentary lifestyle, physical activity, and types of exercise as well as dietary intake of various nutrients or minerals on metabolic parameters conducive to health and prevention of disease with a focus on cardiovascular function.  Preclinical and clinical studies are welcome.

This new information will provide health care professionals with a widespread, clear and update evidence physical activity and dietary intake on cardiometabolic health in the population. 

Dr. Noreen F. Rossi
Dr. Donal S. O'Leary
Guest Editors

Manuscript Submission Information

Manuscripts should be submitted online at www.mdpi.com by registering and logging in to this website. Once you are registered, click here to go to the submission form. Manuscripts can be submitted until the deadline. All submissions that pass pre-check are peer-reviewed. Accepted papers will be published continuously in the journal (as soon as accepted) and will be listed together on the special issue website. Research articles, review articles as well as short communications are invited. For planned papers, a title and short abstract (about 100 words) can be sent to the Editorial Office for announcement on this website.

Submitted manuscripts should not have been published previously, nor be under consideration for publication elsewhere (except conference proceedings papers). All manuscripts are thoroughly refereed through a single-blind peer-review process. A guide for authors and other relevant information for submission of manuscripts is available on the Instructions for Authors page. Nutrients is an international peer-reviewed open access semimonthly journal published by MDPI.

Please visit the Instructions for Authors page before submitting a manuscript. The Article Processing Charge (APC) for publication in this open access journal is 2900 CHF (Swiss Francs). Submitted papers should be well formatted and use good English. Authors may use MDPI's English editing service prior to publication or during author revisions.

Keywords

  • carbohydrates
  • fructose
  • lipid metabolism
  • metabolic intermediates
  • dietary salt intake
  • exercise
  • physical activity
  • blood pressure
  • cardiac function
  • vascular compliance

Benefits of Publishing in a Special Issue

  • Ease of navigation: Grouping papers by topic helps scholars navigate broad scope journals more efficiently.
  • Greater discoverability: Special Issues support the reach and impact of scientific research. Articles in Special Issues are more discoverable and cited more frequently.
  • Expansion of research network: Special Issues facilitate connections among authors, fostering scientific collaborations.
  • External promotion: Articles in Special Issues are often promoted through the journal's social media, increasing their visibility.
  • e-Book format: Special Issues with more than 10 articles can be published as dedicated e-books, ensuring wide and rapid dissemination.

Further information on MDPI's Special Issue policies can be found here.

Published Papers (1 paper)

Order results
Result details
Select all
Export citation of selected articles as:

Research

20 pages, 3882 KiB  
Article
Moderate-Intensity and High-Intensity Interval Exercise Training Offer Equal Cardioprotection, with Different Mechanisms, during the Development of Type 2 Diabetes in Rats
by Sarah D’Haese, Lisa Claes, Iris de Laat, Sven Van Campenhout, Dorien Deluyker, Ellen Heeren, Sibren Haesen, Ivo Lambrichts, Kristiaan Wouters, Casper G. Schalkwijk, Dominique Hansen, BO Eijnde and Virginie Bito
Nutrients 2024, 16(3), 431; https://doi.org/10.3390/nu16030431 - 31 Jan 2024
Cited by 4 | Viewed by 3115
Abstract
Endurance exercise training is a promising cardioprotective strategy in type 2 diabetes mellitus (T2DM), but the impact of its intensity is not clear. We aimed to investigate whether and how isocaloric moderate-intensity exercise training (MIT) and high-intensity interval exercise training (HIIT) could prevent [...] Read more.
Endurance exercise training is a promising cardioprotective strategy in type 2 diabetes mellitus (T2DM), but the impact of its intensity is not clear. We aimed to investigate whether and how isocaloric moderate-intensity exercise training (MIT) and high-intensity interval exercise training (HIIT) could prevent the adverse cardiac remodeling and dysfunction that develop T2DM in rats. Male rats received a Western diet (WD) to induce T2DM and underwent a sedentary lifestyle (n = 7), MIT (n = 7) or HIIT (n = 8). Insulin resistance was defined as the HOMA-IR value. Cardiac function was assessed with left ventricular (LV) echocardiography and invasive hemodynamics. A qPCR and histology of LV tissue unraveled underlying mechanisms. We found that MIT and HIIT halted T2DM development compared to in sedentary WD rats (p < 0.05). Both interventions prevented increases in LV end-systolic pressure, wall thickness and interstitial collagen content (p < 0.05). In LV tissue, HIIT tended to upregulate the gene expression of an ROS-generating enzyme (NOX4), while both modalities increased proinflammatory macrophage markers and cytokines (CD86, TNF-α, IL-1β; p < 0.05). HIIT promoted antioxidant and dicarbonyl defense systems (SOD2, glyoxalase 1; p < 0.05) whereas MIT elevated anti-inflammatory macrophage marker expression (CD206, CD163; p < 0.01). We conclude that both MIT and HIIT limit WD-induced T2DM with diastolic dysfunction and pathological LV hypertrophy, possibly using different adaptive mechanisms. Full article
(This article belongs to the Special Issue Impact of Physical Activity, Diet Quality on Cardiometabolic Health)
Show Figures

Figure 1

Back to TopTop