GLP-1 and GIP Receptor Agonists, Cardiometabolic Disease, Oxidative Stress and Dementia: Novel Insights and Challenges
Special Issue Editor
Interests: endothelial function; arterial stiffness; left ventricular myocardial function; atherosclerosis; oxidative stress; diabetes; cardiometabolic disease
Special Issues, Collections and Topics in MDPI journals
Special Issue Information
Dear Colleagues,
Cardiometabolic disease, namely metabolic disorders such as obesity and type 2 diabetes coupled with hypertension, atherosclerotic cardiovascular disease, heart failure, chronic kidney disease, and metabolic dysfunction-associated steatotic liver disease, has been linked to increased dementia risk. This association is mediated by shared metabolic, inflammatory, and several other neurobiological processes that contribute to cognitive decline. Glucagon-like peptide-1 (GLP-1) and glucose-dependent insulinotropic polypeptide (GIP) receptor agonists show significant promise as a novel therapeutic approach for neurodegenerative diseases, including Alzheimer's disease, Parkinson's disease, and other cognitive disorders, by targeting cardiometabolic dysfunction, reducing oxidative stress and neuroinflammation, and improving cognitive function.
This Special Issue aims to highlight the latest pharmacological advancements and evidence-based clinical strategies for the optimal management of cardiometabolic disease and dementia syndromes.
We invite clinicians, researchers, and experts in cardiovascular medicine, internal medicine, endocrinology, and neurology to contribute original research articles, narrative and systematic reviews, or meta-analyses that focus on new insights into the beneficial mechanisms of GLP-1 and GIP receptor agonists on cardiometabolic disease and cognitive impairment, enhancing patient care and outcomes in these interconnected conditions.
Dr. George Pavlidis
Guest Editor
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Keywords
- glucagon-like peptide-1
- glucose-dependent insulinotropic polypeptide
- cardiometabolic disease
- diabetes
- hypertension
- stroke
- metabolic dysfunction-associated steatotic liver disease
- oxidative stress
- dementia
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