Developing the Coupling or De-Coupling of Osteoclastogenesis vs. Osteogenesis at the Osteoimmune Interface
A special issue of International Journal of Molecular Sciences (ISSN 1422-0067). This special issue belongs to the section "Molecular Immunology".
Deadline for manuscript submissions: 15 July 2026 | Viewed by 888
Special Issue Editor
2. Laboratory of Molecular Microbial Immunity, Division of Periodontology, The Eastman Institute for Oral Health (EIOH), School of Medicine & Dentistry, University of Rochester, Rochester, NY 14620, USA
3. For the Elder & Young’s Center of Oral Medicine Investigation (FEYLOMI), Kaohsiung, Taiwan
Interests: periodontology; oral microbiology and immunology
Special Issue Information
Dear Colleagues,
There have been great advancements in recent years in the osteoimmunology field, whose dawn began a quarter of a century ago. Several innovative breakthroughs linking the underpinning complex interactions and crosstalk at the osteoimmune or mucosae–mesenchymal interface have hinged, bridged, and transcended the early ontogenetic and/or subsequent cascades involving the coupling vs. de-coupling of osteoclastogenesis and osteogenesis towards bone resorption, the formation or repairs/regeneration, etc., consequentially leading to the pathophysiological conditions for homeostasis vs. pathogenesis associated with health or diseases. This Special Issue welcomes the submission of reviews, original research, and short communications, based on the Information for Authors instruction on the IJMS’s webpage (https://www.mdpi.com/authors). With this Special Issue, we aim to provide a platform for new trends, as outlined and showcased, but not limited to, below:
Main topics of interest:
- Cellular or molecular crosstalk associated with coupling or de-coupling of osteoclastogenesis vs. osteogenesis at the osteoimmune interface;
- Specific microenvironment at the osteoimmune interface favoring (or disfavoring) the overall progression of osteoclastogenic pathways;
- Mesenchymal stromal vs. stem cells and hematopoietic progenitors during osteoclastogenesis in bone homeostasis and/or related pathologies;
- Biomaterials or bio-mimics favoring (or disfavoring) the pathways leading to the (de)coupling of osteoclastogenesis and/or osteogenesis;
- Development of ontogenetic lineages (i.e., progenitors vs. precursors associated with hematopoietic or myeloid lineages of monocytes, macrophages, dendritic cells, neutrophils, erythro-megakaryocytes, etc.), consequently leading to osteoclastogenesis and/or osteogenesis.
Dr. Andy Yen-Tung Teng
Guest Editor
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Keywords
- bone and immune cells
- osteoclasts vs. osteoblasts
- interactions between osteoclasts and osteoblasts
- osteoimmune interactions in health and/or disease
- developmental lineages or pathways of osteoclastogenesis
- coupling and/or de-coupling (or crosstalk) for osteoclastogenesis and/or osteogenesis
- biomaterials and/or bio-mimics for bone genesis and/or loss
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