The Immune Escape Mechanisms of Mycobacterium tuberculosis
A special issue of Cells (ISSN 2073-4409). This special issue belongs to the section "Cellular Immunology".
Deadline for manuscript submissions: closed (30 November 2022) | Viewed by 4117
Special Issue Editor
Special Issue Information
Dear Colleagues,
The inability of the immune system to protect the host from the pathogenic effects of M. tuberculosis depends on many factors; however, two main ones can be highlighted: immune system defects at various levels and modulation of M. tuberculosis immune response that allows mycobacteria to survive, multiply, and cause a disease. As an intracellular pathogen, M. tuberculosis adapts to unfavorable external environments and not only modulates its metabolism but also affects the metabolism of the infected host cells. Understanding these metabolic changes should provide insights invaluable for the development of new approaches to tuberculosis treatment.
In this Special Issue of Cells, I invite you to contribute original research articles, reviews, or shorter perspective articles on all aspects related to the theme of “The Immune Escape Mechanisms of Mycobacterium tuberculosis”. Expert articles describing molecular, functional, cellular, biochemical, or general aspects of M. tuberculosis immune evasion are welcome. Relevant topics include but are not limited to:
- Immune modulation
- Dormancy
- Phagosomal rupture
- Cell signaling
- Small RNAs
- Cytokine signaling
- Chemokine function
- In vitro and in vivo models
- Immunopathology of TB
- Inflammation
- Translational medicine
Dr. Vladimir V. Yeremeev
Guest Editor
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