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Feature Paper in Section ‘Cancer Epidemiology and Prevention’ in 2025
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Feature Paper in Section ‘Cancer Epidemiology and Prevention’ in 2025

A special issue of Cancers (ISSN 2072-6694). This special issue belongs to the section "Cancer Epidemiology and Prevention".

Deadline for manuscript submissions: 31 December 2025 | Viewed by 2928

Special Issue Editors

Prof. Dr. Roger Anderson

E-Mail Website
Guest Editor
1. Department of Public Health Sciences, School of Medicine, University of Virginia, Charlottesville, VA 22908, USA
2. Cancer Center, University of Virginia, Charlottesville, VA 22908, USA
Interests: cancer prevention; health services research; health disparities; rural health
Special Issues, Collections and Topics in MDPI journals
Dr. Paweł Koczkodaj

E-Mail Website
Guest Editor
Cancer Epidemiology and Primary Prevention Department, Maria Sklodowska-Curie National Research Institute of Oncology, 02-781 Warsaw, Poland
Interests: cancer prevention; cancer epidemiology; health education; public health
Special Issues, Collections and Topics in MDPI journals

Special Issue Information

Dear Colleagues,

This collection is the second edition of the Special Issue “Feature Paper in Section “Cancer Epidemiology and Prevention” in 2022–2023”

Cancer prevention and cancer epidemiology are among the most crucial elements of oncology. Primary and secondary cancer prevention give efficient tools aiming at reducing risk factor exposure, as well as allowing us to detect cancer at a very early stage. It has been estimated that at least 50% of all cancer cases could be prevented if we follow healthy lifestyle recommendations. Moreover, cancer epidemiology gives us a broad picture on how efficient cancer prevention is and where further improvement is needed. Thanks to cancer epidemiology, we can also observe changes in health behaviors, cancer incidence, and treatment efficacy as a result of policies.

We would like to invite you to submit papers (original and reviews) thematically connected to the scope of this Special Issue. We are particularly, but not exclusively, interested in papers focused on the effects of policies, community programs, and the implementation of new practices on cancer prevention. We believe that Cancers is an ideal platform to exchange perspectives and to present results of studies on cancer prevention and epidemiology.

Prof. Dr. Roger Anderson
Dr. Paweł Koczkodaj
Guest Editors

Manuscript Submission Information

Manuscripts should be submitted online at www.mdpi.com by registering and logging in to this website. Once you are registered, click here to go to the submission form. Manuscripts can be submitted until the deadline. All submissions that pass pre-check are peer-reviewed. Accepted papers will be published continuously in the journal (as soon as accepted) and will be listed together on the special issue website. Research articles, review articles as well as communications are invited. For planned papers, a title and short abstract (about 100 words) can be sent to the Editorial Office for announcement on this website.

Submitted manuscripts should not have been published previously, nor be under consideration for publication elsewhere (except conference proceedings papers). All manuscripts are thoroughly refereed through a single-blind peer-review process. A guide for authors and other relevant information for submission of manuscripts is available on the Instructions for Authors page. Cancers is an international peer-reviewed open access semimonthly journal published by MDPI.

Please visit the Instructions for Authors page before submitting a manuscript. The Article Processing Charge (APC) for publication in this open access journal is 2900 CHF (Swiss Francs). Submitted papers should be well formatted and use good English. Authors may use MDPI's English editing service prior to publication or during author revisions.

Keywords

  • cancer prevention
  • cancer epidemiology
  • health policy
  • public health
  • global health
  • health education
  • health promotion

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Further information on MDPI's Special Issue policies can be found here.

Published Papers (3 papers)

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Review

22 pages, 699 KB  
Review
Causes of Childhood Cancer: A Literature Review (2014–2021)—Part 3: Environmental and Occupational Factors
by Rebecca T. Emeny, Mary E. Butow, Linda Titus, Angela M. Ricci, Pamela J. Bagley, Heather B. Blunt, Alexandra Morgan, Jennifer A. Alford-Teaster, Raymond R. Walston III and Judy R. Rees
Cancers 2025, 17(21), 3516; https://doi.org/10.3390/cancers17213516 - 31 Oct 2025
Viewed by 311
Abstract
Purpose: To achieve a better understanding of the environmental factors that contribute to childhood cancers, so as to inform future prevention efforts. Methods: We conducted a comprehensive review of epidemiological studies on environmental risk factors and childhood cancer, which was published between January [...] Read more.
Purpose: To achieve a better understanding of the environmental factors that contribute to childhood cancers, so as to inform future prevention efforts. Methods: We conducted a comprehensive review of epidemiological studies on environmental risk factors and childhood cancer, which was published between January 2014 and March 2021. Potential exposure sources presented in this review include air pollution, radiation, and parental occupational exposures. We considered exposures during childhood and parental exposures occurring before the child’s conception and during pregnancy in relation to all types of childhood cancer. Results: Aggregated evidence is strong for associations between leukemia and parental/child exposure to traffic pollution, indoor paints, residential pesticides, and parental occupational/nonoccupational exposure to benzene. Evidence is also strong for associations between brain cancer and residential pesticides and parental occupational exposure to agricultural pesticides. Evidence of associations between leukemia and ionizing radiation from radon and nuclear power plants remains mixed, as does evidence of a link between electromagnetic fields (EMFs) and childhood leukemia. Conclusions: Clear associations have been demonstrated between childhood cancer and environmental factors, including parent/child exposure to traffic pollution, occupational/nonoccupational benzene, indoor paints, residential pesticides, and parental occupational exposure to agricultural pesticides. These associations can be used to inform further study of interventions and public health campaigns to reduce risk. Full article
(This article belongs to the Special Issue Feature Paper in Section ‘Cancer Epidemiology and Prevention’ in 2025)
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28 pages, 1745 KB  
Review
Testosterone and Androgen Receptor in Cancers with Significant Sex Dimorphism in Incidence Rates and Survival
by Jianjian Lin, Jingwen Zhu, Jay Fowke, Ramesh Narayanan and Feng Liu-Smith
Cancers 2025, 17(21), 3414; https://doi.org/10.3390/cancers17213414 - 23 Oct 2025
Viewed by 361
Abstract
Several major cancer types exhibit significant sex dimorphism in incidence and survival. Whether and how sex as a biological factor impacts tumorigenesis, progression, and survival warrants full investigation, as such knowledge may lead to novel, precise prevention and treatment strategies. We reviewed epidemiological [...] Read more.
Several major cancer types exhibit significant sex dimorphism in incidence and survival. Whether and how sex as a biological factor impacts tumorigenesis, progression, and survival warrants full investigation, as such knowledge may lead to novel, precise prevention and treatment strategies. We reviewed epidemiological and molecular data on sex differences in cancers of the esophagus, bladder, head and neck, lung, liver, kidney, stomach, and skin melanoma, as well as the potential role of androgens and androgen receptor (AR) activity in these cancers. The potential molecular mechanisms are briefly discussed. Elevated testosterone (T) levels seemed to be associated with increased liver cancer and cutaneous melanoma incidences, and with reduced esophageal cancer risk. AR activity does not always correlate with T levels in tumorigenesis and progression. Higher AR expressions are associated with poorer survival in ESCC, whereas the role of AR in the survival of HNSCC and melanoma patients is inconsistent. The molecular impact of AR in liver cancer, kidney cancer, melanoma, and lung cancer is controversial. However, AR is likely to promote tumor growth and/or progression in esophagus, bladder, head and neck, and stomach cancers, and thus is associated with poor survival. Patients diagnosed with a tumor in this latter group could potentially benefit from therapeutic approaches targeting AR. Overall, the research on sex hormone androgens and AR in these cancers is limited. Further research is needed to determine a possible U-shaped relationship of T with cancer risk, and to decipher the role of testosterone and AR in some of these tumors to facilitate our understanding of sex dimorphism and to explore novel T/AR-based treatment options. Full article
(This article belongs to the Special Issue Feature Paper in Section ‘Cancer Epidemiology and Prevention’ in 2025)
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Figure 1

18 pages, 305 KB  
Review
Causes of Childhood Cancer: A Review of Literature (2014–2021): Part 2—Pregnancy and Birth-Related Factors
by Rebecca T. Emeny, Angela M. Ricci, Linda Titus, Alexandra Morgan, Pamela J. Bagley, Heather B. Blunt, Mary E. Butow, Jennifer A. Alford-Teaster, Raymond R. Walston III and Judy R. Rees
Cancers 2025, 17(15), 2499; https://doi.org/10.3390/cancers17152499 - 29 Jul 2025
Cited by 1 | Viewed by 2047
Abstract
Purpose: To review parental pre-pregnancy and pregnancy exposures in relation to pediatric cancer (diagnosis before age 20). Methods: We conducted literature searches using Ovid Medline and Scopus to find primary research studies, review articles, and meta-analyses published from 2014 to 17 March 2021. [...] Read more.
Purpose: To review parental pre-pregnancy and pregnancy exposures in relation to pediatric cancer (diagnosis before age 20). Methods: We conducted literature searches using Ovid Medline and Scopus to find primary research studies, review articles, and meta-analyses published from 2014 to 17 March 2021. Results: Strong evidence links increased risk of childhood cancer with maternal diabetes, age, and alcohol and coffee consumption during pregnancy. Both paternal and maternal cigarette smoking before and during pregnancy are associated with childhood cancers. Diethylstilbestrol (DES) exposure in utero has long been known to be causally associated with increased risk of vaginal/cervical cancers in adolescent girls. More recent evidence implicates in utero DES exposure to testicular cancer in young men and possible intergenerational effects on ovarian cancer in the granddaughters of women exposed to DES during pregnancy. There is strong evidence that childhood cancer risk is also associated with both high and very low birth weight and with gestational age. Evidence is also strong for the protective effects of maternal vitamin consumption and a healthy diet during pregnancy. Unlike early studies, those reviewed here show no association between in utero exposure to medical ionizing radiation, which may be explained by reductions over time in radiation doses, avoidance of radiation during pregnancy, and/or by inadequate statistical power to detect small increases in risk, rather than a lack of causal association. Evidence is mixed or conflicting for an association between childhood cancer and maternal obesity, birth order, cesarean/instrumental delivery, and prenatal exposure to diagnostic medical radiation. Evidence is weak or absent for associations between childhood cancer and multiple gestations or assisted reproductive therapies, as well as prenatal exposure to hormones other than DES, and medications. Full article
(This article belongs to the Special Issue Feature Paper in Section ‘Cancer Epidemiology and Prevention’ in 2025)
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