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Opinion

How Lazy Reading and Semantic Sloppiness May Harm Progress in Synucleinopathy Research

by 1,2
1
Institut des Maladies Neurodégénératives, UMR 5293, Université de Bordeaux, 33076 Bordeaux, France
2
CNRS, Institut des Maladies Neurodégénératives, UMR 5293, 33076 Bordeaux, France
Academic Editor: Prakash Kulkarni
Biomolecules 2022, 12(2), 228; https://doi.org/10.3390/biom12020228
Received: 22 December 2021 / Revised: 7 January 2022 / Accepted: 26 January 2022 / Published: 28 January 2022
While confronted with the increasing complexity of the neurobiology of Parkinson’s disease (PD), we face the ever-increasing sloppiness of the conceptual definitions associated with poor methodological characterizations and the use of unacknowledged proxies, all of which are harmful contributors to the overall slow progress of PD research. In this opinion paper, I share part of my frustration, acknowledge how I participate in this trend, and propose a simple remedy. Fighting against semantic or conceptual sloppiness is of paramount importance, notably for the benefit of newcomers to the field who otherwise would take for granted the classic assertions found ad nauseam in the literature. View Full-Text
Keywords: Parkinson’s disease; α-synuclein; amyloid; aggregation Parkinson’s disease; α-synuclein; amyloid; aggregation
MDPI and ACS Style

Bezard, E. How Lazy Reading and Semantic Sloppiness May Harm Progress in Synucleinopathy Research. Biomolecules 2022, 12, 228. https://doi.org/10.3390/biom12020228

AMA Style

Bezard E. How Lazy Reading and Semantic Sloppiness May Harm Progress in Synucleinopathy Research. Biomolecules. 2022; 12(2):228. https://doi.org/10.3390/biom12020228

Chicago/Turabian Style

Bezard, Erwan. 2022. "How Lazy Reading and Semantic Sloppiness May Harm Progress in Synucleinopathy Research" Biomolecules 12, no. 2: 228. https://doi.org/10.3390/biom12020228

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