Special Issue "Advances in Pathogenesis and Treatment of Glomerulonephritis"

A special issue of Medicina (ISSN 1648-9144). This special issue belongs to the section "Nephrology".

Deadline for manuscript submissions: 31 August 2023 | Viewed by 712

Special Issue Editor

Dr. Giovanni Conti
E-Mail Website
Guest Editor
Pediatric Nephrology Unit, AOU Policlinic “G Martino”, University of Messina, 98125 Messina, Italy
Interests: pediatric nephrology; pediatric rheumatology; clinical nephrology; glomerulonephritis; nephrotic syndrome; rare diseases; autoimmune diseases; autoinflammatory diseases
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Special Issue Information

Dear Colleagues,

Glomerulonephritis is the main cause of rapid evolution towards chronic renal failure, in case of non-diagnosis and treatment. Glomerulonephritis can be primary or secondary to various systemic diseases, in particular vasculitis and connective tissue diseases. Its pathogenesis is therefore, in the majority of cases, immunological. In recent years, there has been progress on the pathogenetic knowledge of various glomerulonephritis: membranous nephropathy, IgA nephropathy, C3 glomerulonephritis and nephrotic syndrome. Further discoveries have occurred in the genetics of hereditary nephritis. This recent pathogenetic knowledge has led to the use of new therapeutic protocols, also making use of biological drugs, with excellent efficacy and few side effects.

This Special Issue will deal with several aspects of prognostic risk factors, pathogenesis and treatment of glomerulonephritis.

Dr. Giovanni Conti
Guest Editor

Manuscript Submission Information

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Keywords

  • glomerulonephritis
  • nephrotic syndrome
  • membranous nephropathy
  • IgA nephropathy
  • hereditary nephritis
  • vasculitis
  • systemic lupus erythematosus
  • pathogenesis
  • therapy

Published Papers (1 paper)

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Review

Review
The Role of NLRP3 Inflammasome in IgA Nephropathy
Medicina 2023, 59(1), 82; https://doi.org/10.3390/medicina59010082 - 30 Dec 2022
Viewed by 571
Abstract
Immunoglobulin A nephropathy (IgAN) is the most common primary glomerular disease worldwide today. The NLRP3 inflammasome is a polyprotein complex and an important participant in inflammation. Accumulating studies have shown that the NLRP3 inflammasome participates in a variety of kidney diseases, including IgAN. [...] Read more.
Immunoglobulin A nephropathy (IgAN) is the most common primary glomerular disease worldwide today. The NLRP3 inflammasome is a polyprotein complex and an important participant in inflammation. Accumulating studies have shown that the NLRP3 inflammasome participates in a variety of kidney diseases, including IgAN. This review focuses on the role of the NLRP3 inflammasome in IgAN and summarizes multiple involved pathways, which may provide novel treatments for IgAN treatment. Full article
(This article belongs to the Special Issue Advances in Pathogenesis and Treatment of Glomerulonephritis)
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