Oxidative Stress and Autophagy in Cancer Cells
A special issue of International Journal of Molecular Sciences (ISSN 1422-0067). This special issue belongs to the section "Molecular Biology".
Deadline for manuscript submissions: closed (20 March 2026) | Viewed by 3890
Special Issue Editor
Special Issue Information
Dear Colleagues,
Oxidative stress is an important regulator in various pathways including apoptosis and autophagy. Much of the evidence indicates that autophagy and oxidative stress are both key, important contributors to tumorigenesis and cancer progression. Thus, a detailed study of the role of ROS and autophagy is needed to understand their role in cancer initiation and progression and to design effective therapies targeting redox regulation and autophagy systems for cancer therapy. Autophagy is a tumor suppressor pathway but can promote cancer cell survival under diverse stress conditions. Similarly, increased ROS has been implicated in tumorigenesis caused by diverse infectious or environmental agents as well as in the maintenance of cancer cell signaling pathways, but it has been suggested to limit metastasis formation. Various drugs, including phytochemicals and small molecules, are presently being investigated in preclinical and clinical studies that attribute their anticancer activity to ROS induction or to the induction of cell autophagy. Consistently, this Special Issue, “Oxidative Stress and Autophagy in Cancer Cells”, will cover a selection of recent research topics and current review articles in the field on the role of oxidative stress and autophagy processes in cancer progression.
Prof. Dr. Abdelhabib Semlali
Guest Editor
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Keywords
- oxidative stress
- autophagy
- apoptosis
- cancer cells
- cancer cell signaling pathways
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