Autophagy and Apoptosis in Mammal Cells
A special issue of International Journal of Molecular Sciences (ISSN 1422-0067). This special issue belongs to the section "Molecular Biology".
Deadline for manuscript submissions: 31 August 2025 | Viewed by 76
Special Issue Editors
Interests: autophagy; autophagy-dependent cell death; autophagy-related (ATG) genes; apoptosis; cancer cells; metabolic reprogramming; programmed cell death
Special Issue Information
Dear Colleagues,
The importance of autophagy or apoptosis in maintaining cellular/tissue homeostasis is widely known. While autophagy eliminates and recycles damaged or dysfunctional cellular components and is generally considered a prosurvival process, apoptosis is a form of programmed, controlled cell death. Autophagy and apoptosis can be triggered by similar stimuli and their molecular pathways share similar proteins, which both indicate crosstalk between autophagy and apoptosis. Therefore, the dysregulation (defined by inhibition, overactivation, or changes in dynamicity) of one or both processes contributes to the pathogenesis of many diseases, including cancer and neurodegenerative disorders, and is a cause of drug resistance.
In this Special Issue, we encourage you to summarize and broaden the knowledge of common pathways, molecules, the importance of autophagy/apoptosis and their relationship, and the consequences of autophagy/apoptosis manipulation in both physiological and pathological conditions. All these may contribute to the development of knowledge surrounding this area.
Both original and review manuscripts (raising new issues) are welcome.
Dr. Kaja Urbańska
Dr. Anna Litwiniuk
Guest Editors
Manuscript Submission Information
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Keywords
- autophagy
- apoptosis
- cellular homeostasis
- intra- and intercellular crosstalk
- autophagic cell death
- programmed cell death
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