Research on Cell Death in Cancer Immunotherapy

Dear Colleagues,

In this Special Issue we will, on the one hand, summarize the current knowledge, including recently gained crucial insights, on the role of cell death in cancer immunotherapy and, on the other, point towards exciting future opportunities for discovery in this highly exciting field. In addition to discussing the role of different death receptor–ligand systems and their role in enabling or, indeed, preventing cancer immunotherapy, we will also discuss how different factors that have recently emerged as decisive in terms of preventing or enabling cancer immunotherapy are thought to exert their impact on the efficacy of cancer immunotherapy. Finally, we will discuss how different modalities of programmed cell death may differentially affect the outcome of cancer immunotherapy.

Thereby, this review series will provide a timely and highly constructive collection of reviews that complements the current literature in this exciting field of tumor immunobiology. We believe it will become a go-to series for anyone interested in grasping the status quo with regard to both current knowledge of and future research directions on the role of cell death in cancer immunotherapy.

Best wishes

Prof. Dr. Henning Walczak
Guest Editor

Manuscript Submission Information

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Submitted manuscripts should not have been published previously, nor be under consideration for publication elsewhere (except conference proceedings papers). All manuscripts are thoroughly refereed through a single-blind peer-review process. A guide for authors and other relevant information for submission of manuscripts is available on the Instructions for Authors page. Cancers is an international peer-reviewed open access semimonthly journal published by MDPI.

Please visit the Instructions for Authors page before submitting a manuscript. The Article Processing Charge (APC) for publication in this open access journal is 2900 CHF (Swiss Francs). Submitted papers should be well formatted and use good English. Authors may use MDPI's English editing service prior to publication or during author revisions.

• TNF
• FasL
• TRAIL
• death receptor–ligand systems
• LUBAC
• RNF31
• cIAP1/2
• TBK1
• IKK
• RIPK1
• RIPK3
• apoptosis
• necroptosis
• pyroptosis
• ferroptosis