Molecular and Biochemical Mechanisms of Endometriosis
A special issue of Biomedicines (ISSN 2227-9059). This special issue belongs to the section "Molecular and Translational Medicine".
Deadline for manuscript submissions: closed (30 June 2024) | Viewed by 6829
Special Issue Editor
Interests: biochemistry; molecular mechanism; oxidative stress; endometriosis
Special Issues, Collections and Topics in MDPI journals
Special Issue Information
Dear Colleagues,
Endometriosis, a gynecological condition commonly observed in women of reproductive age, is histologically characterized by the presence and growth of endometrial-like glands and stroma outside the uterine cavity and musculature which undergo cyclic proliferation and breakdown similar to that which occurs in the endometrium. This internal bleeding, which cannot leave the body and remains on site, often results in local inflammatory reactions, causing scar tissue formation and adhesions during repair processes. The local microenvironment plays a pivotal role in the onset and progression of an endometriotic lesion, as misplaced endometrial cells need to respond to local stimuli. A very recent proteomic study of peritoneal endometriotic stromal cells revealed extensive metabolic reprogramming and acquisition of cancer-like changes reflected in increased cellular invasiveness and adhesiveness, reduced apoptotic potential, and altered immune function. In this Special Issue entitled, “Molecular and Biochemical Mechanisms of Endometriosis”, we are seeking novel research or review articles highlighting the molecular mechanisms of endometriosis.
Dr. Roberta Fusco
Guest Editor
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Keywords
- biochemistry
- molecular pathways
- endometriosis
- in vivo
- in vitro
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