Antimicrobial Resistance, Epidemiology, Clinical Impact and Molecular Characterization of Multidrug-Resistant ESKAPEE Pathogens
A special issue of Antibiotics (ISSN 2079-6382). This special issue belongs to the section "Antibiotic Therapy in Infectious Diseases".
Deadline for manuscript submissions: 30 April 2026 | Viewed by 341
Special Issue Editors
Interests: microbiology, SARS-CoV-2, molecular diagnostics; molecular typing; vaccines; immunology; mass spectrometry MALDI-TOF
Special Issues, Collections and Topics in MDPI journals
Interests: microbiology; Bacillus anthracis; anthrax; molecular diagnostics; mass spectrometry MALDI-TOF
Special Issues, Collections and Topics in MDPI journals
Special Issue Information
Dear Colleagues,
The acronym ESKAPEE identifies bacteria (Enterococcus faecium, Staphylococcus aureus, Klebsiella pneumoniae, Acinetobacter baumannii, Pseudomonas aeruginosa, Enterobacter spp. and Escherichia coli) that are able to escape the biocidal action of antimicrobials, presenting high rates of multi-resistance. Indeed, commonly available treatment options, including broad-spectrum beta-lactams, carbapenems, glycopeptides, fluoroquinolones, and aminoglycosides, are currently increasingly ineffective against ESKAPEE pathogens; this is predisposing us to a future in which antibiotics are ineffective.
For this reason, ESKAPEE pathogens are responsible for serious nosocomial infections, difficult to fight, worldwide.
In the food sector, ESKAPEE pathogens can contaminate multiple compartments of the food production chain and pose a significant threat to public health. In addition, the overuse or misuse of antibiotics in animal husbandry could increase resistance in these bacteria, thus promoting their spread throughout food production and consumption.
Given the current importance and concern around this topic, this Special Issue is seeking manuscripts that advance our understanding of antimicrobial resistance, virulence, and epidemiology of ESKAPEE pathogens.
Specifically, we are seeking manuscripts in the following areas:
- Mechanisms of antimicrobial resistance ;
- Phenotypic and genotypic assessment of antibiotic resistance;
- Molecular characterization of multidrug-resistant ESKAPEE pathogens;
- Nosocomial outbreaks caused by ESKAPEE pathogens;
- Identification of ESKAPEE pathogens in food and environmental samples;
- Novel drug combinations designed to combat infections caused by ESKAPEE pathogens.
Dr. Lorenzo Pace
Dr. Viviana Manzulli
Guest Editors
Manuscript Submission Information
Manuscripts should be submitted online at www.mdpi.com by registering and logging in to this website. Once you are registered, click here to go to the submission form. Manuscripts can be submitted until the deadline. All submissions that pass pre-check are peer-reviewed. Accepted papers will be published continuously in the journal (as soon as accepted) and will be listed together on the special issue website. Research articles, review articles as well as short communications are invited. For planned papers, a title and short abstract (about 100 words) can be sent to the Editorial Office for announcement on this website.
Submitted manuscripts should not have been published previously, nor be under consideration for publication elsewhere (except conference proceedings papers). All manuscripts are thoroughly refereed through a single-blind peer-review process. A guide for authors and other relevant information for submission of manuscripts is available on the Instructions for Authors page. Antibiotics is an international peer-reviewed open access monthly journal published by MDPI.
Please visit the Instructions for Authors page before submitting a manuscript. The Article Processing Charge (APC) for publication in this open access journal is 2900 CHF (Swiss Francs). Submitted papers should be well formatted and use good English. Authors may use MDPI's English editing service prior to publication or during author revisions.
Keywords
- ESKAPEE
- antimicrobial resistance
- antibiotic resistance
- multidrug-resistant pathogens
- nosocomial infection
- molecular mechanism
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