Order Article Reprints

Gaikwad AV, Eapen MS, Dey S, Bhattarai P, Shahzad AM, Chia C, Jaffar J, Westall G, Sutherland D, Singhera GK, et al. TGF-β1, pSmad-2/3, Smad-7, and β-Catenin Are Augmented in the Pulmonary Arteries from Patients with Idiopathic Pulmonary Fibrosis (IPF): Role in Driving Endothelial-to-Mesenchymal Transition (EndMT). Journal of Clinical Medicine. 2024; 13(4):1160. https://doi.org/10.3390/jcm13041160

Gaikwad, Archana Vijay, Mathew Suji Eapen, Surajit Dey, Prem Bhattarai, Affan Mahmood Shahzad, Collin Chia, Jade Jaffar, Glen Westall, Darren Sutherland, Gurpreet Kaur Singhera, and et al. 2024. "TGF-β1, pSmad-2/3, Smad-7, and β-Catenin Are Augmented in the Pulmonary Arteries from Patients with Idiopathic Pulmonary Fibrosis (IPF): Role in Driving Endothelial-to-Mesenchymal Transition (EndMT)" Journal of Clinical Medicine 13, no. 4: 1160. https://doi.org/10.3390/jcm13041160

Gaikwad, A. V., Eapen, M. S., Dey, S., Bhattarai, P., Shahzad, A. M., Chia, C., Jaffar, J., Westall, G., Sutherland, D., Singhera, G. K., Hackett, T.-L., Lu, W., & Sohal, S. S. (2024). TGF-β1, pSmad-2/3, Smad-7, and β-Catenin Are Augmented in the Pulmonary Arteries from Patients with Idiopathic Pulmonary Fibrosis (IPF): Role in Driving Endothelial-to-Mesenchymal Transition (EndMT). Journal of Clinical Medicine, 13(4), 1160. https://doi.org/10.3390/jcm13041160