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Pathogens
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22 December 2025

Alterations in the Immune Response in Individuals with Latent Tuberculosis Infection

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1
Department of Mathematics and Computer Science, Saint Petersburg State University, 199034 Saint Petersburg, Russia
2
Almazov National Medical Research Center of the Ministry of Health of the Russian Federation, 2, Akkuratov Str., 197341 Saint Petersburg, Russia
3
Medicine Department, Bashkir State Medical University, 450008 Ufa, Russia
4
Institute of Experimental Medicine, 197376 Saint Petersburg, Russia
This article belongs to the Special Issue Innate Immune Response and Pathogen Dynamics

Abstract

Latent tuberculosis infection (LTBI) represents a biologically active yet clinically asymptomatic stage of Mycobacterium tuberculosis (Mtb) persistence. This condition is characterized by subtle immunometabolic alterations reflecting the host–pathogen equilibrium. Understanding the mechanisms and biomarkers associated with the preclinical phase of LTBI is crucial for preventing progression to active tuberculosis (ATB). Recent advances have identified multiple immunological, transcriptomic, metabolic, and imaging-based approaches that enable stratification of individuals at increased risk of LTBI reactivation. Quantitative assays such as IGRA, multiplex and T-cell activation marker (TAM) tests, as well as interferon-related transcriptional signatures, demonstrate predictive potential when combined with functional assays (MGIA) and metabolic imaging (PET/CT). Experimental primate models faithfully reproduce the spectrum from latency to reactivation, allowing for validation of biomarkers and vaccine or immunomodulatory strategies. The review also highlights the particular challenges of multidrug-resistant LTBI (MDR-LTBI), where standard chemoprophylaxis is less effective and immune control plays a decisive role. The preclinical phase of LTBI constitutes a key point in the TB control cascade. Integrating immunological, transcriptomic, and radiological data into risk-based screening algorithms could substantially improve early detection and targeted prevention. Translating research-derived signatures into clinically applicable, standardized, and cost-effective diagnostic tools requires coordinated international efforts, technological transfer, and policy-level support to reduce TB reactivation and transmission, including MDR-TB.

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