The authors wish to make the following correction to this paper [1]: In the published version, Figure 3b appeared as a duplication of Figure 3a, while the figure legend correctly described Figure 3b with different content.
The original version of Figure 3 that has to be corrected is:
Figure 3.
Immunohistochemical staining of CuFi-8 air–liquid-interface cultures. (a) Scheme 8. cells and HBoV-positive lung tumor biopsies, whereas mock-infected CuFi-8 cultures, as well as HBoV-negative lung tumors, were CEA-negative. CuFi-1 and Cufi-5 cells were not CEA-positive at all. (b) PAS–Alcian blue staining reveals higher production of acid mucins in CuFi-8 cells compared to those in CuFi-1 and CuFi-5 cells in general. Beyond that, there is an increased expression of acid mucins after HBoV infection in CuFi-8 cells.
Figure 3.
Immunohistochemical staining of CuFi-8 air–liquid-interface cultures. (a) Scheme 8. cells and HBoV-positive lung tumor biopsies, whereas mock-infected CuFi-8 cultures, as well as HBoV-negative lung tumors, were CEA-negative. CuFi-1 and Cufi-5 cells were not CEA-positive at all. (b) PAS–Alcian blue staining reveals higher production of acid mucins in CuFi-8 cells compared to those in CuFi-1 and CuFi-5 cells in general. Beyond that, there is an increased expression of acid mucins after HBoV infection in CuFi-8 cells.
It should be replaced with the following Figure 3:
Figure 3.
Immunohistochemical staining of CuFi-8 air–liquid-interface cultures. (a) Scheme 8. cells and HBoV-positive lung tumor biopsies, whereas mock-infected CuFi-8 cultures, as well as HBoV-negative lung tumors, were CEA-negative. CuFi-1 and Cufi-5 cells were not CEA-positive at all. (b) PAS–Alcian blue staining reveals higher production of acid mucins in CuFi-8 cells compared to those in CuFi-1 and CuFi-5 cells in general. Beyond that, there is an increased expression of acid mucins after HBoV infection in CuFi-8 cells.
We stress that these errors were purely due to human error and oversight; all corrections done do not affect or change the written proportion of the figure legend, interpretation of the results, or the final conclusions of this manuscript. The manuscript will be updated. The authors would like to apologize for any inconvenience caused. All changes have been reviewed and verified by the Academic Editors.
Author Contributions
M.P. performed cell culturing, extraction of nucleic acids, generation of infectious virus and infection of air–liquid interface cultures. S.K. performed immunohistochemistry and standard histological stainings and analyzed the respective results. W.H.A. performed electron microscopy and analyzed the respective results. V.S. and O.S. analyzed the transcriptome data, performed the IPA analyses, interpreted the data, supervised the entire study, and wrote the manuscript. All authors have read and agreed to the published version of the manuscript.
Funding
This work was supported by an intramural research grant from the Private University of Witten/Herdecke to V.S., a research grant from the Beatrix-Lichtken-Stiftung (Cologne, Germany) to O.S., and a research grant from the Lörcher-Stiftung (Cologne/Frechen, Germany) to O.S.
Conflicts of Interest
The authors declare no conflict of interest.
Reference
- Schildgen, V.; Pieper, M.; Khalfaoui, S.; Arnold, W.H.; Schildgen, O. Human Bocavirus Infection of Permanent Cells Differentiated to Air-Liquid Interface Cultures Activates Transcription of Pathways Involved in Tumorigenesis. Cancers 2018, 10, 410. [Google Scholar] [CrossRef] [PubMed]
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