Kotani, T.; Ikemoto, M.; Matsuda, S.; Masutani, R.; Takeuchi, T.
Human MIKO-1, a Hybrid Protein That Regulates Macrophage Function, Suppresses Lung Fibrosis in a Mouse Model of Bleomycin-Induced Interstitial Lung Disease. Int. J. Mol. Sci. 2022, 23, 9669.
https://doi.org/10.3390/ijms23179669
AMA Style
Kotani T, Ikemoto M, Matsuda S, Masutani R, Takeuchi T.
Human MIKO-1, a Hybrid Protein That Regulates Macrophage Function, Suppresses Lung Fibrosis in a Mouse Model of Bleomycin-Induced Interstitial Lung Disease. International Journal of Molecular Sciences. 2022; 23(17):9669.
https://doi.org/10.3390/ijms23179669
Chicago/Turabian Style
Kotani, Takuya, Masaki Ikemoto, Shogo Matsuda, Ryota Masutani, and Tohru Takeuchi.
2022. "Human MIKO-1, a Hybrid Protein That Regulates Macrophage Function, Suppresses Lung Fibrosis in a Mouse Model of Bleomycin-Induced Interstitial Lung Disease" International Journal of Molecular Sciences 23, no. 17: 9669.
https://doi.org/10.3390/ijms23179669
APA Style
Kotani, T., Ikemoto, M., Matsuda, S., Masutani, R., & Takeuchi, T.
(2022). Human MIKO-1, a Hybrid Protein That Regulates Macrophage Function, Suppresses Lung Fibrosis in a Mouse Model of Bleomycin-Induced Interstitial Lung Disease. International Journal of Molecular Sciences, 23(17), 9669.
https://doi.org/10.3390/ijms23179669
