Molecular Mechanism and Pathway in Residual Cardiovascular Risk
A special issue of International Journal of Molecular Sciences (ISSN 1422-0067). This special issue belongs to the section "Molecular Biology".
Deadline for manuscript submissions: closed (31 August 2021) | Viewed by 445
Special Issue Editor
Interests: thrombogenicity; glucose; platelet; coagulation; inflammation; East Asian paradox
Special Issue Information
Dear Colleagues,
Despite contemporary evidence-based pharmacologic treatments and interventional strategies, clinical events derived from atherosclerotic cardiovascular disease (ASCVD) remain substantially frequent. Studies have focused on controlling the molecules and pathways related to residual CV risks. In addition, the phenotypes of residual CV risks have become more important than before, since therapeutic strategies for controlling these risks are emerging, and several pharmacologic candidates could reduce the risk of CV events. However, there are unmet needs to develop reliable clinical biomarkers (based on molecular mechanisms and pathways) for targeting the right patients with the right therapies at the right time. Therefore, this Special Issue of the International Journal of Molecular Science will cover various emerging targets and important aspects of residual CV risks, which may contribute to the improvement of patient care.
Dr. Young-Hoon Jeong
Guest Editor
Manuscript Submission Information
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Keywords
- Inflammatory pathway: inflammasomes, adipose tissue, and macrophages/monocytes
- Clonal hematopoiesis
- Crosstalk between inflammation and coagulation
- Platelet biology
- Coagulation pathway
- Fibrinolytic pathway
- Lipid risks: cholesterol, triglycerides, and Lp(a)
- Glucagon-like peptide-1 (GLP-1) receptor
- Sodium glucose co-transporter-2 (SGLT-2)
