Molecular Mechanism of Xenobiotics on Respiratory Disorders
A special issue of International Journal of Molecular Sciences (ISSN 1422-0067). This special issue belongs to the section "Molecular Biology".
Deadline for manuscript submissions: 31 July 2026 | Viewed by 162
Special Issue Editor
Interests: genomic medicine and environmental toxicology; cancer genetics and epigenetics; environmental pollution and cancer; air pollution and cancer
Special Issue Information
Dear Colleagues,
Environmental xenobiotics represent an important risk to human health. Compounds such as heavy metals, pesticides, air pollutants, and nano- and microplastics have their origin in anthropogenic activities. The constant increase in industrial and agricultural activities, vehicle emissions, and household products represents a challenge in human health. Cellular homeostasis is constantly compromised by xenobiotics, contributing to a range of respiratory disorders, such as asthma, chronic obstructive pulmonary disease (COPD), and lung cancer, among others. Emerging evidence indicates that genetic and epigenetic alterations, such as DNA damage, aberrant DNA methylation-histone modifications, and altered expression of non-coding ribonucleic acids (ncRNAs), are consequences of the toxic effects of environmental xenobiotics. Moreover, chronic exposure to these compounds during pregnancy and early postnatal stages increases susceptibility to respiratory disorders. Because new epidemiological and toxicological evidence is increasing the scope of environmental health research, the aim of the Special Issue is to explore the relationship between environmental xenobiotics and alterations in genetic and epigenetic mechanisms, focusing on how these changes impact the respiratory system and, eventually, compromise human health.
Dr. Miguel Santibáñez-Andrade
Guest Editor
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Keywords
- xenobiotics
- polycyclic aromatic hydrocarbons-PHAs
- volatile organic compounds-VOCs
- metals
- respiratory disorders
- asthma
- chronic obstructive pulmonary disease (COPD)
- lung cancer
- environmental pollutants
- DNA methylation
- non-coding RNA deregulation
- oxidative damage
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