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Vorwerk J, Liu L, Stadler TH, Frank D, Ahmed HMM, Patnana PK, Kebenko M, Dazert E, Opalka B, von Bubnoff N, et al. Germline Single-Nucleotide Polymorphism GFI1-36N Causes Alterations in Mitochondrial Metabolism and Leads to Increased ROS-Mediated DNA Damage in a Murine Model of Human Acute Myeloid Leukemia. Biomedicines. 2025; 13(1):107. https://doi.org/10.3390/biomedicines13010107

Vorwerk, Jan, Longlong Liu, Theresa Helene Stadler, Daria Frank, Helal Mohammed Mohammed Ahmed, Pradeep Kumar Patnana, Maxim Kebenko, Eva Dazert, Bertram Opalka, Nikolas von Bubnoff, and et al. 2025. "Germline Single-Nucleotide Polymorphism GFI1-36N Causes Alterations in Mitochondrial Metabolism and Leads to Increased ROS-Mediated DNA Damage in a Murine Model of Human Acute Myeloid Leukemia" Biomedicines 13, no. 1: 107. https://doi.org/10.3390/biomedicines13010107

Vorwerk, J., Liu, L., Stadler, T. H., Frank, D., Ahmed, H. M. M., Patnana, P. K., Kebenko, M., Dazert, E., Opalka, B., von Bubnoff, N., & Khandanpour, C. (2025). Germline Single-Nucleotide Polymorphism GFI1-36N Causes Alterations in Mitochondrial Metabolism and Leads to Increased ROS-Mediated DNA Damage in a Murine Model of Human Acute Myeloid Leukemia. Biomedicines, 13(1), 107. https://doi.org/10.3390/biomedicines13010107