Order Article Reprints

Chang J-C, Chang H-S, Chao Y-C, Huang C-S, Lin C-H, Wu Z-S, Chang H-J, Liu C-S, Chuang C-S. Formoterol Acting via β2-Adrenoreceptor Restores Mitochondrial Dysfunction Caused by Parkinson’s Disease-Related UQCRC1 Mutation and Improves Mitochondrial Homeostasis Including Dynamic and Transport. Biology. 2024; 13(4):231. https://doi.org/10.3390/biology13040231

Chang, Jui-Chih, Huei-Shin Chang, Yi-Chun Chao, Ching-Shan Huang, Chin-Hsien Lin, Zhong-Sheng Wu, Hui-Ju Chang, Chin-San Liu, and Chieh-Sen Chuang. 2024. "Formoterol Acting via β2-Adrenoreceptor Restores Mitochondrial Dysfunction Caused by Parkinson’s Disease-Related UQCRC1 Mutation and Improves Mitochondrial Homeostasis Including Dynamic and Transport" Biology 13, no. 4: 231. https://doi.org/10.3390/biology13040231

Chang, J.-C., Chang, H.-S., Chao, Y.-C., Huang, C.-S., Lin, C.-H., Wu, Z.-S., Chang, H.-J., Liu, C.-S., & Chuang, C.-S. (2024). Formoterol Acting via β2-Adrenoreceptor Restores Mitochondrial Dysfunction Caused by Parkinson’s Disease-Related UQCRC1 Mutation and Improves Mitochondrial Homeostasis Including Dynamic and Transport. Biology, 13(4), 231. https://doi.org/10.3390/biology13040231