Leal, N.S.; Dentoni, G.; Schreiner, B.; Naia, L.; Piras, A.; Graff, C.; Cattaneo, A.; Meli, G.; Hamasaki, M.; Nilsson, P.;
et al. Amyloid β-Peptide Increases Mitochondria-Endoplasmic Reticulum Contact Altering Mitochondrial Function and Autophagosome Formation in Alzheimer’s Disease-Related Models. Cells 2020, 9, 2552.
https://doi.org/10.3390/cells9122552
AMA Style
Leal NS, Dentoni G, Schreiner B, Naia L, Piras A, Graff C, Cattaneo A, Meli G, Hamasaki M, Nilsson P,
et al. Amyloid β-Peptide Increases Mitochondria-Endoplasmic Reticulum Contact Altering Mitochondrial Function and Autophagosome Formation in Alzheimer’s Disease-Related Models. Cells. 2020; 9(12):2552.
https://doi.org/10.3390/cells9122552
Chicago/Turabian Style
Leal, Nuno Santos, Giacomo Dentoni, Bernadette Schreiner, Luana Naia, Antonio Piras, Caroline Graff, Antonio Cattaneo, Giovanni Meli, Maho Hamasaki, Per Nilsson,
and et al. 2020. "Amyloid β-Peptide Increases Mitochondria-Endoplasmic Reticulum Contact Altering Mitochondrial Function and Autophagosome Formation in Alzheimer’s Disease-Related Models" Cells 9, no. 12: 2552.
https://doi.org/10.3390/cells9122552
APA Style
Leal, N. S., Dentoni, G., Schreiner, B., Naia, L., Piras, A., Graff, C., Cattaneo, A., Meli, G., Hamasaki, M., Nilsson, P., & Ankarcrona, M.
(2020). Amyloid β-Peptide Increases Mitochondria-Endoplasmic Reticulum Contact Altering Mitochondrial Function and Autophagosome Formation in Alzheimer’s Disease-Related Models. Cells, 9(12), 2552.
https://doi.org/10.3390/cells9122552