Yakhnitsa, V.; Thompson, J.; Ponomareva, O.; Ji, G.; Kiritoshi, T.; Mahimainathan, L.; Molehin, D.; Pruitt, K.; Neugebauer, V.
Dysfunction of Small-Conductance Ca2+-Activated Potassium (SK) Channels Drives Amygdala Hyperexcitability and Neuropathic Pain Behaviors: Involvement of Epigenetic Mechanisms. Cells 2024, 13, 1055.
https://doi.org/10.3390/cells13121055
AMA Style
Yakhnitsa V, Thompson J, Ponomareva O, Ji G, Kiritoshi T, Mahimainathan L, Molehin D, Pruitt K, Neugebauer V.
Dysfunction of Small-Conductance Ca2+-Activated Potassium (SK) Channels Drives Amygdala Hyperexcitability and Neuropathic Pain Behaviors: Involvement of Epigenetic Mechanisms. Cells. 2024; 13(12):1055.
https://doi.org/10.3390/cells13121055
Chicago/Turabian Style
Yakhnitsa, Vadim, Jeremy Thompson, Olga Ponomareva, Guangchen Ji, Takaki Kiritoshi, Lenin Mahimainathan, Deborah Molehin, Kevin Pruitt, and Volker Neugebauer.
2024. "Dysfunction of Small-Conductance Ca2+-Activated Potassium (SK) Channels Drives Amygdala Hyperexcitability and Neuropathic Pain Behaviors: Involvement of Epigenetic Mechanisms" Cells 13, no. 12: 1055.
https://doi.org/10.3390/cells13121055
APA Style
Yakhnitsa, V., Thompson, J., Ponomareva, O., Ji, G., Kiritoshi, T., Mahimainathan, L., Molehin, D., Pruitt, K., & Neugebauer, V.
(2024). Dysfunction of Small-Conductance Ca2+-Activated Potassium (SK) Channels Drives Amygdala Hyperexcitability and Neuropathic Pain Behaviors: Involvement of Epigenetic Mechanisms. Cells, 13(12), 1055.
https://doi.org/10.3390/cells13121055