Erdő-Bonyár, S.; Rapp, J.; Szinger, D.; Minier, T.; Kumánovics, G.; Czirják, L.; Berki, T.; Simon, D.
Ligation of TLR Homologue CD180 of B Cells Activates the PI3K/Akt/mTOR Pathway in Systemic Sclerosis and Induces a Pathological Shift in the Expression of BAFF Receptors. Int. J. Mol. Sci. 2022, 23, 6777.
https://doi.org/10.3390/ijms23126777
AMA Style
Erdő-Bonyár S, Rapp J, Szinger D, Minier T, Kumánovics G, Czirják L, Berki T, Simon D.
Ligation of TLR Homologue CD180 of B Cells Activates the PI3K/Akt/mTOR Pathway in Systemic Sclerosis and Induces a Pathological Shift in the Expression of BAFF Receptors. International Journal of Molecular Sciences. 2022; 23(12):6777.
https://doi.org/10.3390/ijms23126777
Chicago/Turabian Style
Erdő-Bonyár, Szabina, Judit Rapp, Dávid Szinger, Tünde Minier, Gábor Kumánovics, László Czirják, Timea Berki, and Diána Simon.
2022. "Ligation of TLR Homologue CD180 of B Cells Activates the PI3K/Akt/mTOR Pathway in Systemic Sclerosis and Induces a Pathological Shift in the Expression of BAFF Receptors" International Journal of Molecular Sciences 23, no. 12: 6777.
https://doi.org/10.3390/ijms23126777
APA Style
Erdő-Bonyár, S., Rapp, J., Szinger, D., Minier, T., Kumánovics, G., Czirják, L., Berki, T., & Simon, D.
(2022). Ligation of TLR Homologue CD180 of B Cells Activates the PI3K/Akt/mTOR Pathway in Systemic Sclerosis and Induces a Pathological Shift in the Expression of BAFF Receptors. International Journal of Molecular Sciences, 23(12), 6777.
https://doi.org/10.3390/ijms23126777