Roles of MicroRNA in Cardiac Remodeling and Heart Failure
A special issue of International Journal of Molecular Sciences (ISSN 1422-0067). This special issue belongs to the section "Molecular Biology".
Deadline for manuscript submissions: 31 May 2026 | Viewed by 5
Special Issue Editor
Special Issue Information
Dear Colleagues,
This Special Issue, "Roles of MicroRNA (miRNA) in Cardiac Remodeling and Heart Failure," is timely, leveraging the heightened global interest in miRNA biology following the anticipated 2025 Nobel Prize. Its central goal is to present a state-of-the-art collection of research addressing how miRNAs regulate the cellular and molecular processes driving pathological cardiac remodeling and the progression to heart failure.
We invite high-quality submissions, including original research papers and review articles, exploring the regulatory roles of miRNAs across the translational spectrum. We particularly welcome contributions from experimental and preclinical studies that elucidate novel miRNA mechanisms involved in precursor cardiovascular conditions like atherosclerosis, myocardial infarction, and hypertension, establishing their critical link to subsequent heart failure and cardiac dysfunction.
Furthermore, the Issue seeks important findings from clinical studies focusing on the diagnostic and prognostic utility of tissue and circulating miRNAs as potential biomarkers for patient risk stratification and personalized therapy. Finally, we also highlight advancements in the design and delivery of miRNA-targeting therapeutic strategies. By integrating the broad pathophysiology of CVDs relevant to heart failure, this collection will serve as an essential resource, charting the future of miRNA-based diagnostics and therapeutics in cardiovascular medicine.
Prof. Dr. Ján Kyselovič
Guest Editor
Manuscript Submission Information
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Keywords
- miRNAs
- cardiac remodeling
- cardiovascular disease
- atherosclerosis
- myocardial infarction
- cardiac dysfunction
- heart failure
- biomarkers
- personalized therapy
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