Molecular Mechanisms of Mitochondrial Autophagy
A special issue of Biomolecules (ISSN 2218-273X). This special issue belongs to the section "Molecular Biology".
Deadline for manuscript submissions: closed (31 December 2021) | Viewed by 736
Special Issue Editor
Interests: mitochondrial autophagy (mitophagy); mitochondrial dynamics; mitochondrial diseases; mitochondrial cristae remodeling; mitochondrial metabolism
Special Issues, Collections and Topics in MDPI journals
Special Issue Information
Dear Colleagues,
The mitochondrion is a double membrane-bound organelle that generates most of the cellular ATP through oxidative phosphorylation (OXPHOS) in the majority of eukaryotic cells. In addition, the mitochondrion plays an essential role in a number of cellular processes. Mitochondrial autophagy (mitophagy) is a conserved cellular process for selectively eliminating dysfunctional or unneeded mitochondria in eukaryotic cells. Mitophagy plays a crucial role in maintaining cellular homeostasis. Defects in mitophagy contribute to a variety of human diseases, such as cancer and neurodegenerative, cardiovascular, and metabolic diseases. The PINK1/Parkin- and FUNDC1-mediated pathways mainly regulate mitophagy. However, the molecular mechanisms of mitophagy under certain cellular stresses remain largely unknown. For this Special Issue, we seek original research or review articles that focus on the molecular mechanisms and signaling pathways of mitophagy under certain physiological conditions, the novel methods detecting mitophagy, and the identification and synthesis of new biomolecules (or small-molecule compounds) regulating mitophagy.
Prof. Dr. Zhiyin Song
Guest Editor
Manuscript Submission Information
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Keywords
- mitophagy
- mitophagy receptor
- autophagy
- PINK1
- Parkin
- FUNDC1
- mitophagy inhibitor
- mitophagy activator
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