Al Otaibi, A.; Al Shaikh Mubarak, S.; Al Hejji, F.; Almasaud, A.; Al Jami, H.; Iqbal, J.; Al Qarni, A.; Harbi, N.K.A.; Bakillah, A.
Thapsigargin and Tunicamycin Block SARS-CoV-2 Entry into Host Cells via Differential Modulation of Unfolded Protein Response (UPR), AKT Signaling, and Apoptosis. Cells 2024, 13, 769.
https://doi.org/10.3390/cells13090769
AMA Style
Al Otaibi A, Al Shaikh Mubarak S, Al Hejji F, Almasaud A, Al Jami H, Iqbal J, Al Qarni A, Harbi NKA, Bakillah A.
Thapsigargin and Tunicamycin Block SARS-CoV-2 Entry into Host Cells via Differential Modulation of Unfolded Protein Response (UPR), AKT Signaling, and Apoptosis. Cells. 2024; 13(9):769.
https://doi.org/10.3390/cells13090769
Chicago/Turabian Style
Al Otaibi, Abeer, Sindiyan Al Shaikh Mubarak, Fatimah Al Hejji, Abdulrahman Almasaud, Haya Al Jami, Jahangir Iqbal, Ali Al Qarni, Naif Khalaf Al Harbi, and Ahmed Bakillah.
2024. "Thapsigargin and Tunicamycin Block SARS-CoV-2 Entry into Host Cells via Differential Modulation of Unfolded Protein Response (UPR), AKT Signaling, and Apoptosis" Cells 13, no. 9: 769.
https://doi.org/10.3390/cells13090769
APA Style
Al Otaibi, A., Al Shaikh Mubarak, S., Al Hejji, F., Almasaud, A., Al Jami, H., Iqbal, J., Al Qarni, A., Harbi, N. K. A., & Bakillah, A.
(2024). Thapsigargin and Tunicamycin Block SARS-CoV-2 Entry into Host Cells via Differential Modulation of Unfolded Protein Response (UPR), AKT Signaling, and Apoptosis. Cells, 13(9), 769.
https://doi.org/10.3390/cells13090769