Hunt, B.G.; Jones, A.; Lester, C.; Davis, J.C.; Benight, N.M.; Waltz, S.E.
RON (MST1R) and HGFL (MST1) Co-Overexpression Supports Breast Tumorigenesis through Autocrine and Paracrine Cellular Crosstalk. Cancers 2022, 14, 2493.
https://doi.org/10.3390/cancers14102493
AMA Style
Hunt BG, Jones A, Lester C, Davis JC, Benight NM, Waltz SE.
RON (MST1R) and HGFL (MST1) Co-Overexpression Supports Breast Tumorigenesis through Autocrine and Paracrine Cellular Crosstalk. Cancers. 2022; 14(10):2493.
https://doi.org/10.3390/cancers14102493
Chicago/Turabian Style
Hunt, Brian G., Angelle Jones, Carissa Lester, James C. Davis, Nancy M. Benight, and Susan E. Waltz.
2022. "RON (MST1R) and HGFL (MST1) Co-Overexpression Supports Breast Tumorigenesis through Autocrine and Paracrine Cellular Crosstalk" Cancers 14, no. 10: 2493.
https://doi.org/10.3390/cancers14102493
APA Style
Hunt, B. G., Jones, A., Lester, C., Davis, J. C., Benight, N. M., & Waltz, S. E.
(2022). RON (MST1R) and HGFL (MST1) Co-Overexpression Supports Breast Tumorigenesis through Autocrine and Paracrine Cellular Crosstalk. Cancers, 14(10), 2493.
https://doi.org/10.3390/cancers14102493