Van Heesbeen, H.J.; Von Oerthel, L.; De Vries, P.M.; Wagemans, C.M.R.J.; Smidt, M.P.
Neuronal Dot1l Activity Acts as a Mitochondrial Gene-Repressor Associated with Human Brain Aging via H3K79 Hypermethylation. Int. J. Mol. Sci. 2023, 24, 1387.
https://doi.org/10.3390/ijms24021387
AMA Style
Van Heesbeen HJ, Von Oerthel L, De Vries PM, Wagemans CMRJ, Smidt MP.
Neuronal Dot1l Activity Acts as a Mitochondrial Gene-Repressor Associated with Human Brain Aging via H3K79 Hypermethylation. International Journal of Molecular Sciences. 2023; 24(2):1387.
https://doi.org/10.3390/ijms24021387
Chicago/Turabian Style
Van Heesbeen, Hendrikus J., Lars Von Oerthel, Paul M. De Vries, Cindy M. R. J. Wagemans, and Marten P. Smidt.
2023. "Neuronal Dot1l Activity Acts as a Mitochondrial Gene-Repressor Associated with Human Brain Aging via H3K79 Hypermethylation" International Journal of Molecular Sciences 24, no. 2: 1387.
https://doi.org/10.3390/ijms24021387
APA Style
Van Heesbeen, H. J., Von Oerthel, L., De Vries, P. M., Wagemans, C. M. R. J., & Smidt, M. P.
(2023). Neuronal Dot1l Activity Acts as a Mitochondrial Gene-Repressor Associated with Human Brain Aging via H3K79 Hypermethylation. International Journal of Molecular Sciences, 24(2), 1387.
https://doi.org/10.3390/ijms24021387