Epstein-Barr Virus Disease Mechanisms and Therapeutic Strategies

Topic Information

Dear Colleagues,

Epstein–Barr virus (EBV) infects a significant percentage (>95%) of the population and establishes a persistent infection. However, most of these infections are asymptomatic. A small but significant number of EBV-positive patients develop a variety of lymphomas, including Burkitt lymphoma, diffuse large-cell B cell lymphoma, classical Hodgkin lymphoma and epithelial malignancies including nasopharyngeal carcinoma and gastric carcinoma. Furthermore, there is increasing evidence that EBV may be a trigger/driver for some autoimmune diseases, including multiple sclerosis, systemic lupus erythematosus, Sjogren’s syndrome and rheumatoid arthritis. Most studies have focused on the potential role of latency-associated gene products in these diseases, but there is accumulating evidence that the products of genes expressed during lytic or abortive-lytic activity may also be involved. This Topic is devoted to exploring the role of how molecules produced during lytic/abortive-lytic replication of EBV may contribute to modifying the host’s immune system in immunocompetent individuals and how the proteins may contribute to the disease process.

Prof. Dr. Marshall Vance Williams
Dr. Jonathan Kerr
Topic Editors

Keywords

Participating Journals

Journal Name	Impact Factor	CiteScore	Launched Year	First Decision (median)	APC
Biomolecules biomolecules	4.8	9.4	2011	18.4 Days	CHF 2700
International Journal of Molecular Sciences ijms	4.9	8.1	2000	16.8 Days	CHF 2900
Pathogens pathogens	3.3	6.4	2012	15.3 Days	CHF 2200
Vaccines vaccines	5.2	8.9	2013	18.6 Days	CHF 2700
Viruses viruses	3.8	7.3	2009	17.1 Days	CHF 2600

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