Animal Models Reveal Immunological and Neurological Toxicity Induced by Environmental Pollutants
Special Issue Editor
Interests: heavy metal; toxicity; immunity; drugs
Special Issues, Collections and Topics in MDPI journals
Special Issue Information
In recent years, environmental pollutants have been recognized as a crucial factor contributing to tissue dysfunction. These pollutants are highly diverse, including pharmaceuticals, antibiotic-resistant bacteria and genes, pesticides, veterinary medicines, personal care products, emerging pollutants like PFAS, microplastics, nanomaterials, heavy metals, radionuclides, nutrients, pathogens, indicator organisms, oil, and hydrocarbons. They can be detected in multiple sources such as air, water, soil, food, and industrial products. The health risks they pose to animals have captured extensive attention.
Recent research has indicated that environmental pollutants can trigger immunological and neurological toxicities. In the immune system, pollutants may disrupt innate immunity, cause inflammation, and impact the overall immunological balance, increasing the susceptibility to infectious diseases. In the nervous system, they can lead to neurodegenerative diseases and other neurological disorders. These toxicities are often associated with the development of various diseases, including cardiovascular diseases, chronic obstructive pulmonary disease, chronic kidney disease, neurodegenerative diseases, and cancer.
Therefore, it is essential to utilize animal models to uncover the immunological and neurological toxicities induced by environmental pollutants. This approach will offer evidence for the treatment and prevention of the harm caused by environmental pollutants.
This Special Issue aims to provide a platform for researchers to explore the molecular mechanisms of environmental pollutant-induced immunological and neurological toxicities and their long-term health impacts, offering new perspectives for the prevention and treatment of related diseases.
Dr. Yanzhu Zhu
Guest Editor
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Keywords
- Neuroscience
- Environmental pollutants
- Inflammation
- Reveal oxidative stress
- Animal models
- Tissue dysfunction
- Toxicology
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